Skip to main content Accessibility help

We use cookies to distinguish you from other users and to provide you with a better experience on our websites. Close this message to accept cookies or find out how to manage your cookie settings.

Close cookie message
×
  1. Home
  2. Search

Refine search

Refine search

Access:
Content type:
Publication date:
Apply
Subject: Show more
Journals Show more
Publishers: Show more
Societies Show more
Series: Show more
Collections: Show more

Actions for selected content:

Select all | Deselect all
  • View selected items
  • Export citations
  • Download PDF (zip)
  • Save to Kindle
  • Save to Dropbox
  • Save to Google Drive

Save Search

You can save your searches here and later view and run them again in "My saved searches".

Please provide a title, maximum of 40 characters.
Cancel
×

11 results

  • Chapter 10.1 - Bone Disorders
  • from 10 - Bone Disorders and Stroke
  • Edited by Derya Uluduz, Istanbul Üniversitesi
  • Anita Arsovska
  • Book: Rare Causes of Stroke
  • Published online: 06 October 2022
  • Print publication: 01 September 2022, pp 336-342
  • View extract

    Summary

    Bone disorders are extremely rare cause of stroke. Several bone disorders and abnormalities have been shown to be associated with stroke: osteoporosis, skull disorders (Osteopetrosis, Paget’s disease, and Craniosynostosis), skeletal abnormalities (Spondyloepiphyseal dysplasia, Schimke syndrome, and Camurati-Engelmann disease), bone tumors (osteochondromas and osteosarcoma), fibrocartilaginous and fat emboli, and orthopedics procedures.The relationship of these diseases with stroke has been shown, but mostly insufficiently elucidated yet. Bone diseases related stroke is highly plausible but only after other, more common causes, are ruled out

  • The Value of Carotid Doppler Ultrasound in Asymptomatic Extracranial Arterial Disease
  • Natan M. Bornstein, Lorraine G. Chadwick, John W. Norris
  • Journal: Canadian Journal of Neurological Sciences / Volume 15 / Issue 4 / November 1988
  • Published online by Cambridge University Press: 18 September 2015, pp. 378-383
    • Article
      • You have access
    • PDF
    • Export citation
  • View abstract

    Carotid Doppler is an accurate, safe and repeatable method of assessing arterial calibre, for distinguishing harmless neck bruits and to identify the stroke prone individual. It is completely non-invasive and can be used serially to monitor progression in carotid stenosis. It is a valuable clinical tool in diagnosis and management in patients at risk of stroke, but has definite limitations, such as in differentiating carotid occlusion from severe stenosis. B-mode imaging, although valuable in identifying arterial anatomy, and detecting plaques, cannot accurately evaluate the degree of stenosis. It is of limited value in identifying plaque hemorrhage and ulceration. Doppler ultrasound technology has advanced rapidly in the last decade, especially in the combination of B-mode imaging and Doppler (Duplex), as well as in evaluating of the intracranial circulation (transcranial Doppler). In the next decade, it may become the new gold standard for evaluating the extracranial and intracranial circulation.

  • Basilar Artery Blood Flow in Subclavian Steal
  • Natan M. Bornstein, Adam Krajewski, John W. Norris
  • Journal: Canadian Journal of Neurological Sciences / Volume 15 / Issue 4 / November 1988
  • Published online by Cambridge University Press: 18 September 2015, pp. 417-419
    • Article
      • You have access
    • PDF
    • Export citation
  • View abstract

    Subclavian “steal”, when blood siphons from one vertebral artery to the other, has been suggested as a cause of brain stem ischaemia and stroke. We investigated 33 patients using transcranial Doppler to determine the direction and velocity of basilar blood flow. All patients had severe subclavian stenosis with reversed vertebral blood flow in the ipsilateral artery previously demonstrated by extracranial Doppler. Basilar flow was normal in direction in all cases, but its velocity was significantly increased (p<0.0008) compared to age- and sex-matched controls. These findings, in conjunction with previous observations using extracranial Doppler techniques, suggest that subclavian steal is little more than a harmless haemodynamic phenomenon.

  • 17 - Management of acute ischemic stroke and its complications
  • from Section IV - Therapeutic strategies and neurorehabilitation
  • Edited by Michael Brainin, Zentrum für Klinische Neurowissenschaften, Donnau-Universität, Krems, Austria, Wolf-Dieter Heiss, Universität zu Köln
  • Book: Textbook of Stroke Medicine
  • Published online: 05 May 2010
  • Print publication: 19 November 2009, pp 243-257
  • View extract

    Summary

    General management of elevated blood pressure, blood glucose and body temperature

    Monitoring the blood pressure (BP), glucose levels and temperature in acute stroke patients is an often neglected matter although it may have an important impact upon the patients' outcome. In the Tel Aviv stroke register, recorded between the years 2001 and 2003, 32% of acute stroke patients in the emergency room had glucose levels higher than 150 mg/dl, higher systolic BP than 140 mmHg was found in 77% of the patients and 17% of patients had temperatures above 37°C on admission. These numbers are representative of other centers as well. This chapter will summarize the current knowledge regarding the management of the above.

    Hypertensive blood pressure values in acute ischemic stroke

    Several observations have demonstrated spontaneous elevation of blood pressure in the first 24–48 hrs after stroke onset with a significant spontaneous decline after a few days [1–3]. Several mechanisms may be responsible for the increased blood pressure, including stress, pain, urinary retention, Cushing effect due to increased intracranial pressure and the activation of the sympathetic, renin–angiotensin and ACTH– cortisol pathways. Despite the increased prevalence of hypertension following stroke, optimal management has not been yet established. Several arguments speak for lowering the elevated BP: risks of hemorrhagic transformation, cerebral edema, recurrence of stroke and hypertensive encephalopathy.

  • 58 - BONE DISORDERS AND CEREBROVASCULAR DISEASES
  • from PART V: - SYSTEMIC DISORDERS THAT ALSO INVOLVE THE CEREBROVASCULAR SYSTEM
  • Edited by Louis R. Caplan, Julien Bogousslavsky
  • Book: Uncommon Causes of Stroke
  • Published online: 06 January 2010
  • Print publication: 09 October 2008, pp 423-428
  • View abstract

    Summary

    The association of elevated hematocrit and fibrinogen levels with increased stroke risk has led to continued interest in hemorheologic factors and their role in the development of vascular disease and acute stroke. Viscosity increases logarithmically at the lowest shear rates and this effect is magnified at higher hematocrit levels. Fibrinogen may play a causal role in ischemic stroke through several mechanisms. The most prominent of these is its essential role in thrombosis, both as the substrate for fibrin clot formation and as a facilitator of platelet aggregation. The risk of stroke in sickle cell disease (SCD) varies according to genotype and is highest in patients with homozygous SS. Plasma hyperviscosity syndromes are treated by plasmapheresis (plasma exchange) to remove the paraproteins and thereby reduce hyperviscosity and hypervolemia. Newer techniques of cell centrifugation, plasma separation, and filtration may also be useful.