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32 - Anterior cerebral artery

from PART II - VASCULAR TOPOGRAPHIC SYNDROMES

Published online by Cambridge University Press:  17 May 2010

John C.M. Brust
Affiliation:
Harlem Hospital Center, NewYork, USA
Seiji Kazui
Affiliation:
National Cardiovascular Center, Osaka, Japan
Julien Bogousslavsky
Affiliation:
Université de Lausanne, Switzerland
Louis R. Caplan
Affiliation:
Harvard Medical School
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Summary

Anatomy

The anterior cerebral artery (ACA) arises as the medial branch of the bifurcation of the internal carotid artery (ICA) at the level of the anterior clinoid process. The ACA supplies the whole of the medial surfaces of the frontal and parietal lobes, the anterior four-fifths of the corpus callosum, the frontobasal cerebral cortex, the anterior diencephalon, and other deep structures. The posterior extent of the ACA depends on the extent of the supply of the posterior cerebral artery and its splenial branches. The band of lateral cortex supplied by the ACA is wider anteriorly, often extending beyond the superior frontal sulcus, and is narrowed progressively posteriorly. The ACA can be divided into five segments (A1–A5) (Fischer, 1938). The A1 segment, also called the proximal ACA, is the part between the internal carotid bifurcation and the anterior communicating artery (ACoA). The postcommunicating part of the ACA comprises the segments A2, A3, A4, and A5, which are generally designated the distal ACA. The A2 and A3 segments together are referred to as the ascending segment, with inferior forward convexity (A2) and superior forward convexity (A3). The A4 and A5 segments form the horizontal segment, extending posteriorly to the coronal suture (A4) and from there to the artery's termination.

Basal arteries and their perforating branches

Many perforating branches arise from the anterior circle of Willis (Table 32.1, Fig. 32.1). Most of them are thin, and only the recurrent artery of Heubner can be easily identified by means of angiography. As with other penetrating arteries supplying the diencephalon and basal ganglia, those originating from the ACA and ACoA have sparse anastomoses (‘end-zone arteries’).

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Stroke Syndromes , pp. 439 - 450
Publisher: Cambridge University Press
Print publication year: 2001

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