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Theory of Schizophrenia: Comments

Published online by Cambridge University Press:  06 August 2018

Einar Kringlen*
Affiliation:
Department of Psychiatry, University of Oslo, Vinderen, Oslo 3, Norway

Abstract

Since Gregory Bateson and his co-workers presented their double-blind theory of the aetiology of schizophrenia in the 1950s, no grand comprehensive theory of the disorder has appeared. The double-blind theory was never accepted by the worldwide psychiatric community, and empirical research during the 1970s gave no support to it. In recent years, psychiatrists have focused attention on limited aspects of the illness, and mini-theories have flourished, but this tendency has been typical for the social sciences in general. Therefore, it is refreshing to have two attempts to integrate biological, psychological, and social viewpoints. But have these succeeded?

Type
Research Article
Copyright
Copyright © 1994 The Royal College of Psychiatrists 

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References

Alanen, Y. O. (1994) An attempt to integrate the individual-psychological and interactional concepts of the origins of schizophrenia. British Journal of Psychiatry, 164 (suppl. 23), 5661.Google Scholar
Ciompi, L. (1988) The Psyche and Schizophrenia: the Bond between Affect and Logic. Cambridge, MA: Harvard University Press.Google Scholar
Ciompi, L. (1991) Affects as central organising and integrating factors: a new psychosocial/biological model of the psyche. British Journal of Psychiatry, 159, 97105.Google Scholar
Ciompi, L. (1994) Affect logic: an integrative model of the psyche and its relations to schizophrenia. British Journal of Psychiatry, 164 (suppl. 23), 5155.Google Scholar
Egeland, J. A., Gerhard, D. S., Pauls, D. L., et al (1987) Bipolar affective disorders linked to DNA-markers on chromosome II. Nature, 325, 783787.Google Scholar
Engel, G. (1977) The need for a new medical model: a challenge for biomedicine. Science, 196, 129136.Google Scholar
Firth, W. J. (1991) Chaos – predicting the unpredictable. British Medical Journal, 303, 15651568.Google Scholar
Kelsoe, J. R., Ginns, E. T., Egeland, J. A., et al (1989) Re-evaluation of the linkage relationship between chromosome II loci and the gene for bipolar affective disorder in the Old Order Amish. Nature, 342, 238243.Google Scholar
Onstad, S., Skre, I., Torgersen, S., et al (1991) Subtypes of schizophrenia – evidence from a twin-family study. Acta Psychiatrica Scandinavica, 84, 203206.CrossRefGoogle ScholarPubMed
Plomin, R. (1986) Development: Genetics and Psychology. Hillsdale, New York: Lawrence Erlbaum.Google Scholar
Tienari, P., Wynne, L. C., Moring, J., et al (1994) The Finnish Adoptive Family Study of Schizophrenia: implications for family research. British Journal of Psychiatry, 164 (suppl. 23), 2026.Google Scholar
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