Hostname: page-component-76fb5796d-wq484 Total loading time: 0 Render date: 2024-04-27T00:47:39.665Z Has data issue: false hasContentIssue false
  • English
  • Français

Harrington Rod Instrumentation: A Cause of Brown-Sequard Syndrome

Published online by Cambridge University Press:  18 September 2015

C.B. Van Orman  and
H.Z. Darwish
C.B. Van Orman
Affiliation:
Division of Neurology, Department of Pediatrics, University of Calgary, Calgary
H.Z. Darwish*
Affiliation:
Division of Neurology, Department of Pediatrics, University of Calgary, Calgary
*
Alberta Children's Hospital, 1820 Richmond Road, Calgary, Alberta, Canada T2T 5C7
Rights & Permissions [Opens in a new window]

Abstract:

Core share and HTML view are not available for this content. However, as you have access to this content, a full PDF is available via the ‘Save PDF’ action button.

We describe a child with Brown-Sequard syndrome beginning at C8-T1, immediately following Harrington rod instrumentation for idiopathic scoliosis. We believe this is most likely related to either a common variation of the branches of the anterior spinal artery to one side or due to rupture of the ipsilateral spinal vein. This possible complication should be considered in patients undergoing Harrington rod instrumentation.

Type
Original Articles
Information
Canadian Journal of Neurological Sciences , Volume 15 , Issue 1 , February 1988 , pp. 44 - 46
Copyright
Copyright © Canadian Neurological Sciences Federation 1988

References

REFERENCES

1.Wynne-Davis, R. Familial (idiopathic) scoliosis: A family survey. J Bone Joint Surg (Br) 1968; 50: 24–3.CrossRefGoogle Scholar
2.Winter, RB. Adolescent idiopathic scoliosis. N Engl J Med 1986; 314: 1379–80.CrossRefGoogle ScholarPubMed
3.Kevin, HA. Fundamentals and basic principles of scoliosis. Orthopaedic Review 1983; 12: 31–40.Google Scholar
4.MacEwen, GD, Bunnell, WP, Sriram, K. Acute neurological complications in the treatment of scoliosis. J Bone Joint Surg (Am) 1975; 57–A: 404–8.CrossRefGoogle ScholarPubMed
5.Wilber, RG, Thompson, GH, Shaffer, JW, et al. Postoperative neurological deficits in segmental spinal instrumentation. J Bone Joint Surg (Br) 1984; 66: 1178–1187.CrossRefGoogle ScholarPubMed
6.Nash, CL, Brown, RH. The intraoperative monitoring of spinal cord function: its growth and current status. Orthop Clin North Am 1979; 1: 919–926.CrossRefGoogle Scholar
7.Lesser, RP, Randzens, P, Luders, H, et al. Postoperative neurological deficits may occur despite unchanged intraoperative somatosensory evoked potentials. Ann Neurol 1986; 19: 22–25.CrossRefGoogle ScholarPubMed
8.Bernard, TN, Johnston, CE, Roberts, JM, et al. Late complications due to wire breakage in segmental spinal instrumentation. J Bone Joint Surg (Am) 1983; 65–A: 1339–1345.CrossRefGoogle ScholarPubMed
9.Hilal, SK, Keim, HA. Selective spinal angiography in adolescent scoliosis. Radiology 1972; 102: 349–359.CrossRefGoogle ScholarPubMed
10.Dolan, EJ, Transfeldt, EE, Tator, CHet al. The effect of spinal distraction on regional spinal cord blood flow in cats. J Neurosurg 1980; 53: 756–764.CrossRefGoogle ScholarPubMed
11.Wells, CEC. Clinical aspects of spinovascular disease. Proc Roy Soc Med 1966; 59: 790–795.CrossRefGoogle ScholarPubMed