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Case 44 - Mimicks of acute thoracic aortic syndromes: aortic dissection, intramural hematoma, and penetrating aortic ulcer

from Section 4 - Cardiovascular

Published online by Cambridge University Press:  05 March 2013

Martin L. Gunn
Affiliation:
University of Washington School of Medicine
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Summary

Imaging description

Acute thoracic aortic dissection is the most common aortic catastrophe. It is caused by a tear along the laminar planes of the media layer of the aortic wall, with formation of a blood-filled channel. The diagnosis of aortic dissection is usually quite simple using contrast-enhanced multi-detector CT angiography (CTA). However, several artifacts can simulate aortic dissection.

Most artifacts lead to a false-positive diagnosis.

Pulsation artifact mimics aortic dissection, particularly Stanford type A aortic dissection. Within the ascending aorta, pulsation artifact occurs principally in the left anterior and right posterior wall of the ascending aorta. To discriminate, look for the artifact extending into the adjacent mediastinal fat, and similar “pseudoflaps” in the adjacent superior vena cava and main pulmonary artery. This can be particularly valuable to discriminate between type A and type B aortic dissection, and can even be performed using prospective gating without beta blockade in the acutely unwell patient (Figure 44.1). Aortic pulsation artifact can obscure subtle intimal irregularities, and impairs assessment of aortic valve and coronary involvement for type A dissections. These relationships are well assessed with gating (Figure 44.2) [1].

Type
Chapter
Information
Pearls and Pitfalls in Emergency Radiology
Variants and Other Difficult Diagnoses
, pp. 146 - 150
Publisher: Cambridge University Press
Print publication year: 2013

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References

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Tittle, SL, Lynch, RJ, Cole, PE, et al. Midterm follow-up of penetrating ulcer and intramural hematoma of the aorta. J Thorac Cardiovasc Surg. 2002;123(6):1051–9.CrossRefGoogle ScholarPubMed
Nathan, DP, Boonn, W, Lai, E, et al. Presentation, complications, and natural history of penetrating atherosclerotic ulcer disease. J Vasc Surg. 2012;55(1):10–15.CrossRefGoogle ScholarPubMed
Tsai, TT, Nienaber, CA, Eagle, KA.Acute aortic syndromes. Circulation. 2005;112(24):3802–13.CrossRefGoogle ScholarPubMed

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