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Imidazolinone resistance in several Amaranthus hybridus populations

Published online by Cambridge University Press:  20 January 2017

Daniel H. Poston
Affiliation:
Eastern Shore Agricultural Research and Extension Center, Virginia Polytechnic Institute and State University, Painter, VA 23420
Thomas E. Hines
Affiliation:
Eastern Shore Agricultural Research and Extension Center, Virginia Polytechnic Institute and State University, Painter, VA 23420

Abstract

Field and greenhouse studies were conducted to evaluate the responses of several imidazolinone (IMI)-resistant Amaranthus hybridus populations to various acetolactate synthase (EC 4.1.3.18)-inhibiting herbicides. In 1996 field studies, IMI resistance was confirmed in one A. hybridus population (R4) that was not cross-resistant to the sulfonylurea herbicides CGA-277476, chlorimuron, or thifensulfuron. Amaranthus hybridus control with triazolopyrimidine herbicides was ≤ 35%, but control with cloransulam-methyl or flumetsulam plus cloransulam-methyl was higher than with IMI herbicides. Follow-up greenhouse studies were conducted in 1997 and 1998 to investigate the response of one IMI-susceptible (S) A. hybridus population collected near Painter, VA, and four IMI-resistant A. hybridus populations (R1, R2, R3, R4) collected from fields in Somerset County, MD, to postemergence imazethapyr, chlorimuron, thifensulfuron, pyrithiobac, and cloransulam-methyl applications. Resistance to imazethapyr was confirmed in all R populations, and no practical level of cross-resistance to chlorimuron, thifensulfuron, pyrithiobac, or cloransulam-methyl was detected. Based on resistance ratios, R populations were slightly more tolerant to chlorimuron and slightly more sensitive to pyrithiobac, thifensulfuron, and cloransulam-methyl than the S population.

Type
Research Article
Copyright
Copyright © Weed Science Society of America 

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Footnotes

Current address: Delta Research and Extension Center, Mississippi State University, Stoneville, MS 38776

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