Skip to main content Accessibility help
×
Hostname: page-component-77c89778f8-fv566 Total loading time: 0 Render date: 2024-07-24T22:21:03.824Z Has data issue: false hasContentIssue false

23 - Human and Animal Bites

from Part IV - Clinical Syndromes – Skin and Lymph Nodes

Published online by Cambridge University Press:  05 March 2013

Ellie J. C. Goldstein
Affiliation:
University of California
David Schlossberg
Affiliation:
Temple University School of Medicine, Philadelphia
Get access

Summary

Annually, in the United States, more than 5 million animal and untold numbers of human bite wounds occur and account for 10 000 persons hospitalized and 1% (300 000) of all emergency department visits. Patients with bite wounds are also commonly seen as outpatients in primary care physician and specialist (orthopedics, plastic/hand surgery, infectious diseases physicians) offices. Some patients will attempt to conceal the nature of the injury with human bite wounds. The bacteriology of these wounds is diverse and comprises oral flora organisms, both aerobic and anaerobic, of the biting animal/human, the victim's skin flora, and occasionally environmental isolates.

ANIMAL BITES

Microbiology

An extensive number of bacterial species are isolated from infected dog and cat bite wounds. Pasteurella species, especially Pasteurella multocida and Pasteurella septica, will be present in 75% and 65% of cat bite and dog bite wounds, respectively. Anaerobes will be present in 50% of dog bite wounds and 67% of cat bite wounds. Streptococci, excluding group A β-hemolytic streptococci (Streptococcus pyogenes) are present in 46% of dog and cat bite wounds, whereas Staphylococcus aureus is present in 20% of dog bites but only 4% of cat bites. Streptococcus pyogenes (group A), if present, usually comes from the victim's skin because it is rarely isolated from dog oral flora. Streptococcus aureus is also a secondary invader originating as skin flora. Capnocytophaga canimorsus is an uncommon wound isolate but has been associated with bacteremia, some fatal, in asplenic and cirrhotic patients.

Type
Chapter
Information
Publisher: Cambridge University Press
Print publication year: 2008

Access options

Get access to the full version of this content by using one of the access options below. (Log in options will check for institutional or personal access. Content may require purchase if you do not have access.)

Save book to Kindle

To save this book to your Kindle, first ensure coreplatform@cambridge.org is added to your Approved Personal Document E-mail List under your Personal Document Settings on the Manage Your Content and Devices page of your Amazon account. Then enter the ‘name’ part of your Kindle email address below. Find out more about saving to your Kindle.

Note you can select to save to either the @free.kindle.com or @kindle.com variations. ‘@free.kindle.com’ emails are free but can only be saved to your device when it is connected to wi-fi. ‘@kindle.com’ emails can be delivered even when you are not connected to wi-fi, but note that service fees apply.

Find out more about the Kindle Personal Document Service.

Available formats
×

Save book to Dropbox

To save content items to your account, please confirm that you agree to abide by our usage policies. If this is the first time you use this feature, you will be asked to authorise Cambridge Core to connect with your account. Find out more about saving content to Dropbox.

Available formats
×

Save book to Google Drive

To save content items to your account, please confirm that you agree to abide by our usage policies. If this is the first time you use this feature, you will be asked to authorise Cambridge Core to connect with your account. Find out more about saving content to Google Drive.

Available formats
×