To send content items to your account,
please confirm that you agree to abide by our usage policies.
If this is the first time you use this feature, you will be asked to authorise Cambridge Core to connect with your account.
Find out more about sending content to .
To send content items to your Kindle, first ensure email@example.com
is added to your Approved Personal Document E-mail List under your Personal Document Settings
on the Manage Your Content and Devices page of your Amazon account. Then enter the ‘name’ part
of your Kindle email address below.
Find out more about sending to your Kindle.
Note you can select to send to either the @free.kindle.com or @kindle.com variations.
‘@free.kindle.com’ emails are free but can only be sent to your device when it is connected to wi-fi.
‘@kindle.com’ emails can be delivered even when you are not connected to wi-fi, but note that service fees apply.
Promotion of regeneration in the injured nervous system
Stephen G. Waxman, Department of Neurology and Center for Neuroscience Research, Yale University School of Medicine, New Haven and Rehabilitation Research Center, VA Connecticut Healthcare System, West Haven, CT, USA
This chapter discusses the organization and function of normal myelinated axons and demyelinated and dysmyelinated axons. The myelinated fiber consists of an axon and its surrounding myelin sheaths. As a result of its high electrical resistance and low capacitance, the myelin functions as an insulator which prevents current loss during action potential conduction. Action potential conduction is rapid, and occurs in a unidirectional manner because sodium channels close soon after activation and remain refractory for a short time. Occasional reports have purported that neuroelectric blocking factors or sodium channel blocking factors may contribute to axonal conduction block in neuro-inflammatory disorders. Oligodendrocyte and Schwann cell-mediated remyelination of central nervous system (CNS) axons can both enhance conduction along CNS axons. Although the relationship of axonal degeneration to demyelination is not yet clear, neuroprotection of axons has emerged as a major theme in recent MS research.
Email your librarian or administrator to recommend adding this to your organisation's collection.