To save content items to your account,
please confirm that you agree to abide by our usage policies.
If this is the first time you use this feature, you will be asked to authorise Cambridge Core to connect with your account.
Find out more about saving content to .
To save content items to your Kindle, first ensure email@example.com
is added to your Approved Personal Document E-mail List under your Personal Document Settings
on the Manage Your Content and Devices page of your Amazon account. Then enter the ‘name’ part
of your Kindle email address below.
Find out more about saving to your Kindle.
Note you can select to save to either the @free.kindle.com or @kindle.com variations.
‘@free.kindle.com’ emails are free but can only be saved to your device when it is connected to wi-fi.
‘@kindle.com’ emails can be delivered even when you are not connected to wi-fi, but note that service fees apply.
This chapter reviews data on disruptions in explicit and implicit memory associated with post-traumatic stress disorder (PTSD) and dissociation in laboratory research. It discusses the several biological and cognitive mechanisms proposed to account for autobiographical memory impairment. PTSD is often associated with increased startle responsiveness during threatening contexts, as well as increases in physiological responsiveness to trauma-related stimuli. Among the mechanisms proposed to account for autobiographical memory deficits for traumatic experiences, three general categories emerge: compromises to brain structures that result in changes in function, cognitive mechanisms, and consequences of post-traumatic symptoms. Autobiographical memory impairment could occur via active or involuntary cognitive processes. Active inhibitory processes contribute to difficulty retrieving trauma-related memories because of repeated inhibition of event-related cues. Associations between autobiographical memory impairments for trauma and PTSD/dissociation symptoms are difficult to reconcile with laboratory findings.