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NMS and genetic drug oxidation

Published online by Cambridge University Press:  02 January 2018

Koichi Otani
Affiliation:
Department of Neuropsychiatry Hirosaki University Hospital Hirosaki, Japan
Sunao Kaneko
Affiliation:
Department of Neuropsychiatry Hirosaki University Hospital Hirosaki, Japan
Yutaka Fukushima
Affiliation:
Department of Neuropsychiatry Hirosaki University Hospital Hirosaki, Japan
Kan Chiba
Affiliation:
Department of Neuropsychiatry Hirosaki University Hospital Hirosaki, Japan
Takashi Ishizaki
Affiliation:
Clinical Research Institute National Medical Center Tokyo, Japan
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Abstract

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Copyright © 1991 The Royal College of Psychiatrists 

References

Bertilsson, L., Henthorn, T. K., Sanz, E., et al (1989) Importance of genetic factors in the regulation of diazepam metabolism: relationship to S-mephenytoin, but not debrisoquin, hydroxylation phenotype. Clinical Pharmacology & Therapeutics, 45, 348355.CrossRefGoogle Scholar
Dahl-Puustinen, M-L., Lidén, A., Alm, C., et al (1989) Disposition of perphenazine is related to polymorphic debrisoquin hydroxylation in human being. Clinical Pharmacology & Therapeutics, 46, 7881.CrossRefGoogle Scholar
Gram, L. F., Debruyne, D., Caillard, V., et al (1989) Substantial rise in sparteine metabolic ratio during haloperidol treatment. British Journal of Clinical Pharmacology, 27, 272275.CrossRefGoogle ScholarPubMed
Horai, Y., Nakano, M., Ishizaki, T., et al (1989) Metoprolol and mephenytoin oxidation polymorphisms in far eastern oriental subjects: Japanese versus mainland Chinese. Clinical Pharmacology & Therapeutics, 46, 198207.CrossRefGoogle ScholarPubMed
Santos, J. L., Cabranes, J. A., Almoguera, J., et al (1989) Clinical implications of determination of plasma haloperidol levels. Acta Psychiatrica Scandinavica, 79, 348354.CrossRefGoogle ScholarPubMed
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