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High Resolution Optical Microscopy Will Play a Major Role in Functional Assessments and the Prevention of Disease

Published online by Cambridge University Press:  02 July 2020

Robert W. Bradford, Chairman
Affiliation:
Bradford Research Institute and Capital University of Integrative Medicine, Chula Vista, California91911
Curriculum Oversight
Affiliation:
Bradford Research Institute and Capital University of Integrative Medicine, Chula Vista, California91911
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Extract

The typical aging processes can be characterized by a gradual alteration and essential breakdown of the functional systems of the body. Numerous factors play important roles in the alterations of these pathways from biochemical individuality and genetic predisposition to environmental insults and deficiency states.1 Clinical research, as but one example, has clearly documented the role of free radicals (ROS) or oxidative injury in disease and aging affecting the body's basic cellular structures.

The objective of functional or health assessments is to be able to detect in vivo stresses and imbalances in the biological systems, while the patient is asymptomatic, so that early therapeutic intervention can resolve the problems prior to disease onset or accelerated aging. The ability to assess risk factors and treat the sub-clinical metabolic toxicities, deficiency states, hormonal imbalances, oxidative injury (ROS), immunodeficiencies, enzyme down regulations, antioxidant status, metal toxicities, cardiovascular stresses, organ reserves/stresses and detox systems in a low-complexity, cost-effective office procedure is true preventive medicine.

Type
Recent Advances in Light Microscopy
Copyright
Copyright © Microscopy Society of America

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References

References:

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2Bradford, RW, Allen, HW, Oxidology, The Study of Reactive Oxygen Toxic Species (ROTS) and Their Metabolism in Health and Disease, Second Edition, 1997. Bradford Research Institute, Chula Vista, CA 91911. Chem Absts. 127:276650 (1997)Google Scholar
3Shacter, E, Williams, JA, Levine, RL, Oxidative Modification of Fibrinogen Inhibits Thrombin-Catalyzed Clot Formation, Free Radic Biol Med 1995; 18:815–21CrossRefGoogle ScholarPubMed