Dietary Restraint Theory

The influential dietary restraint theory proposed by Herman and Mack (Reference Herman and Mack1975) emphasises the role of dieting as a precipitating factor in the maintenance of binge eating, suggesting that restricting intake to the point of becoming chronically hungry made people more susceptible to overeating. Polivy and Herman (Reference Polivy and Herman1985) presented evidence from the case study literature and animal studies that indicated that food restriction or dieting preceded the first binge-eating episode. On the basis of this evidence, they proposed that it is restrictive dieting that causes people to commence and continue binge eating. Polivy and Herman further suggested that people who binge eat are trapped in a cycle of restricting and then bingeing and then restricting, and so on. They postulated that people who engaged in restricted eating were then cognitively regulating their eating (as they were no longer relying on the physiological cues to eat). These individuals tended to engage in a ‘black-and-white’ thinking style in relation to food and eating whereby strict rules were applied dictating what foods were able to be eaten, in what quantities, and when. In doing so, they believed that this made the individual vulnerable to disinhibition, particularly when one of these strict rules was broken, which consequentially led to binge-eating episodes due to their ‘all or nothing’ cognitive style (Polivy & Herman, Reference Polivy and Herman1985). Evidence that supports dietary restraint theory comes from prospective studies showing self-reported dieting could reliably predict future onset of binge eating in both non-clinical samples (Stice & Agras, Reference Stice and Agras1998; Stice, Killen, Hayward, & Taylor, Reference Stice, Killen, Hayward and Taylor1998; Stice, Presnell, & Spangler, Reference Stice, Presnell and Spangler2002) and samples of eating-disorder patients (Killen et al., Reference Killen, Taylor, Hayward, Haydel, Wilson, Hammer and Strachowski1996; Stice, Reference Stice2002). Further evidence in support of the dietary restraint theory comes from a series of lab-based experiments that examine the phenomenon of counter-regulation (eating more after consuming a calorically rich food or meal) in restrained eaters (Polivy, Reference Polivy1996; Herman & Polivy, Reference Herman, Polivy, Pirke, Ploog and Vandereycken1988; Herman, Polivy, & Esses, Reference Herman, Polivy and Esses1987; Knight & Boland, Reference Knight and Boland1989; Polivy, Heatherton & Herman, Reference Polivy, Heatherton and Herman1988). They measured disinhibited eating by calculating the amount of food (e.g., ice cream) consumed in a ‘taste-testing’ experiment, where participants were randomly allocated to consume a preload (i.e., a snack or meal high in calories such as a chocolate milkshake) prior to the ‘taste test’. Overall, the results of these experiments show that non-dieters tend to regulate their intake and consume less food after a calorically high preload; however, dieters were found to eat more following the consumption of the preload relative to no preload eating, demonstrating a counter-regulatory response. In addition, Federoff, Polivy, and Herman (Reference Fedoroff, Polivy and Herman1997, Reference Fedoroff, Polivy and Herman2003) compared the effect of food cues on desire to eat and cravings for food in restrained eaters versus unrestrained eaters. The results of this study found that restrained eaters were more responsive to food cues than unrestrained eaters; the restrained eaters ate significantly more of the cued food than the unrestrained eaters.

The dietary restraint model is commonly criticised for being too limited in its explanation of binge-eating pathology (Waller, Reference Waller, Fairburn and Brownell2002; Waters, Hill, & Waller, Reference Waters, Hill and Waller2001; Wilson, Reference Wilson, Fairburn and Brownell2002). Although the evidence suggests that dietary restriction does play an important role in triggering and maintaining binge-eating behaviour, this model does not provide an explanation for binge episodes experienced by those who have not engaged in dietary restriction (McManus & Waller, Reference McManus and Waller1995). To understand and effectively treat binge eating, it is necessary to develop a more comprehensive model of the disordered behaviour that considers psychological as well as behavioural/environmental factors.

Cognitive Models of Binge Eating

Garner and Bemis (Reference Garner and Bemis1982) provided an early cognitive model for AN that had its basis in Beck's cognitive model of depression (Beck, Reference Beck1979). Garner and Bemis’ theory places emphasis on the individual having distorted cognitions, such as the belief ‘I must become thin’. They describe predisposing characteristics of AN patients as being introverted, emotionally sensitive, high achievers, with perfectionistic standards. They suggest that these individuals become withdrawn and depressed, typically triggered by an external situation. They then suggest that the individual develops the belief, through family or society and the media, that losing weight will alleviate their distress, and so they begin engaging in behaviours such as dietary restriction or exercise in order to achieve this. Garner and Bemis propose that the initial success of weight loss leads to feelings of increased self-control, and the behaviour is reinforced by positive comments by others. This serves to reinforce their belief that ‘thinness is paramount’. The individual then develops a fear of gaining weight, and as a result becomes preoccupied with losing weight and maintaining strict control over their eating increases. As a consequence, the disorder becomes self-maintaining (Garner & Bemis, Reference Garner and Bemis1982). Therefore, Garner and Bemis propose that, as in Beck's cognitive model of depression (Beck, Reference Beck1979), distorted cognitions and beliefs are key maintaining factors in AN. Garner and Bemis’ cognitive model of AN, together with the dietary restraint model, provided the theoretical framework for many of the major relevant cognitive theories of binge eating, including the widely cited cognitive-behavioural model of BN proposed by Fairburn, Cooper, and Cooper (Reference Fairburn, Cooper, Cooper, Brownell and Foreyt1986).

Cognitive-Behavioural Model of BN

Fairburn et al. (Reference Fairburn, Cooper, Cooper, Brownell and Foreyt1986) extended the ideas of Garner and Bemis’ (Reference Garner and Bemis1982) model for AN to also account for BN, emphasising the importance of body shape and weight as a key maintaining factor, and thoughts about food and eating as secondary to these (M. Cooper, Cohen-Tovée, Todd, Wells, & Tovée, Reference Cooper, Cohen-Tovée, Todd, Wells and Tovée1997; Fairburn, Reference Fairburn, Fairburn and Brownell2002a). Fairburn et al. described the cognitive processes present in BN to be very similar to those proposed for AN: a pervasive fear of fatness; the over-valuation of thinness and self-control; and a rigid, dichotomous style of thinking. This model emphasised the tendency for these individuals to judge their self-worth in relation to their eating, shape, and weight, and their control over these factors. This style of self-evaluation was thought to be paired with perfectionism, low self-esteem, and negative self-appraisals. Fairburn et al. propose that these beliefs and values about weight and shape lead to the observed behaviours of restricting caloric intake and either increased frequency or avoidance of weighing. Therefore, this model accounts for the binge-eating episodes that occur for individuals with BN as a consequence of their rigid, dichotomous thinking with regard to their eating. Their extreme concern about their weight and shape leads them to adopt strict and usually unrealistic rules about eating. Fairburn et al. proposed that when these individuals deviate from their self-imposed rules, they believe they have ‘failed’, ‘lack self-control’, and are ‘weak’. As a result, Fairburn et al. posit that they abandon all control over their eating and commence a binge-eating episode. The binge-eating episodes are said to maintain the central cognitive disturbance of BN by acting to magnify the individual's concerns about their ability to control their eating, weight, and shape. This, in turn, is thought to further reinforce their engagement in dietary restraint and thereby increase the chance of future binge episodes. Refer to Figure 1 for a representation of Fairburn et al.’s model of BN. Thus, both the dietary restraint model as described by Herman and Mack (Reference Herman and Mack1975) and Fairburn et al.’s cognitive model for BN (Fairburn et al., Reference Fairburn, Cooper, Cooper, Brownell and Foreyt1986) present a vicious cycle whereby dietary restriction (driven by strict rules about eating) and binge-eating episodes (triggered when a dietary rule has been broken and/or chronic hunger as a result of the restriction) drive the disordered eating habits and related compensatory behaviours of those with BN, mediated by beliefs about eating, weight, shape, and their control.

Note: Reproduced from Fairburn, Cooper, and Shafran (Reference Fairburn, Cooper and Shafran2003) with permission from the publisher.

FIGURE 1 The cognitive behavioural theory of bulimia nervosa.

In addition to the components of the cognitive-behavioural model described above, Fairburn has also acknowledged that binge episodes are particularly likely to occur at times of low mood, with the emotional state thought to undermine the individual's ability to maintain their strict control over their eating (Fairburn, Reference Fairburn, Fairburn and Brownell2002a; Z. Cooper & Fairburn, Reference Cooper and Fairburn2011). Fairburn proposed a role of binge eating in neutralising these negative mood states, suggesting that individuals were using binge eating as a form of mood modulation (Fairburn, Reference Fairburn, Fairburn and Brownell2002a). Fairburn has also highlighted that the role of compensatory behaviours such as self-induced vomiting, misuse of laxatives or diuretics, or intense exercise following a binge, suggesting that individuals with BN use compensatory behaviours to minimise the risk of weight gain following a binge episode (Fairburn, Reference Fairburn, Fairburn and Brownell2002a). As a result, one of the main barriers against overeating is removed and individuals become stuck in a vicious cycle of bingeing and purging, which further lowers self-esteem and further reinforces the binge-purge cycle (Fairburn, Reference Fairburn, Fairburn and Brownell2002a).

Fairburn, Marcus, and Wilson (Reference Fairburn, Marcus, Wilson, Fairburn and Wilson1993) developed a cognitive-behavioural treatment program for BN (CBT-BN) based on the cognitive-behavioural model of binge eating. The National Institute for Clinical Excellence guidelines (NICE, 2004, as cited in Wilson & Shafran, Reference Wilson and Shafran2005) named this approach as the recommended first-line treatment for individuals with BN. Subsequently, this treatment approach has been found remission rates for binge eating in ~30–50% of cases (Wilson et al., Reference Wilson, Grilo and Vitousek2007). Agras, Walsh, Fairburn, Wilson, and Kraemer (Reference Agras, Walsh, Fairburn, Wilson and Kraemer2000) compared CBT treatment with interpersonal psychotherapy (IPT), a treatment originally developed for depression that has been modified for the treatment of BN (Fairburn, Reference Fairburn, Fairburn and Brownell2002b). Agras et al. completed a randomised control trial of 220 BN patients who received treatment over a 20-week period with a follow-up at 1 year post-treatment. At the end of the 20-week treatment, results found CBT to be superior to IPT at eliminating binge eating. However, at the 1-year, post-treatment follow-up, there were no statistically significant differences in binge-eating abstinence between CBT and IPT where patients treated with CBT had largely maintained their post-treatment gains and those with IPT had seen a continued reduction in their binge-eating frequency during the follow-up period (Agras et al., Reference Agras, Walsh, Fairburn, Wilson and Kraemer2000). The results of this study demonstrated that CBT treatment provided a faster reduction in binge-eating behaviour compared to IPT, which is suggested to be due to the targeted nature of CBT. Agras et al. concluded that this result indicates a non-shared mechanism of action between these two treatments. Interestingly, despite observing reduced binge-eating frequency as a consequence of these two treatments, the core psychopathology of BN, such as shape and weight concerns, were found not to have been significantly reduced at the end of treatment (Agras et al., Reference Agras, Walsh, Fairburn, Wilson and Kraemer2000). A follow-up study (Wilson, Fairburn, Agras, Walsh, & Kraemer, Reference Wilson, Fairburn, Agras, Walsh and Kraemer2002) that investigated the efficacy of the CBT treatment for BN found that a decrease in dietary restraint mediated the reduction in binge eating. They also found that mid-treatment measures of self-efficacy regarding eating behaviour, negative affect, and body shape and weight were also significantly associated with post-treatment outcome at 20 weeks (Wilson et al., Reference Wilson, Fairburn, Agras, Walsh and Kraemer2002).

Although CBT-BN demonstrated some good outcomes, with 30–50% of BN patients treated with CBT having ceased binge eating and purging completely, a significant proportion remained symptomatic at the end of treatment (Wilson et al., Reference Wilson, Fairburn, Agras, Walsh and Kraemer2002). When modified for the treatment of BED, this CBT approach has demonstrated 79% binge abstinence rates at post-treatment, superior to IPT (73%; Wilfley et al., Reference Wilfley, Welch, Stein, Spurrell, Cohen, Saelens and Matt2002). But again, there was no difference between CBT and IPT in the long-term maintenance of change, with approximately 40% of participants still engaging in binge eating at the 12-month follow-up: 41% and 39% for CBT and IPT respectively (Wilfley et al., Reference Wilfley, Welch, Stein, Spurrell, Cohen, Saelens and Matt2002). These less than ideal results indicate that CBT treatment, and the model that it is based on, requires further development, with the aim of appropriately understanding and addressing the factors that continue to maintain binge-eating pathology in approximately half the cases in these clinical populations.

Escape Theory of Binge Eating

Providing an alternative to dietary restraint theory and instead focusing on the link between negative affect and binge eating, Heatherton & Baumeister (Reference Heatherton and Baumeister1991) proposed the escape theory of binge eating. In this theory, they suggest that individuals who binge eat do so in order to escape from self-awareness. They explain that these individuals hold high personal expectations about their weight and shape. They wish to be perceived favourably by others, are self-conscious, and assume that others are judging them. As a result, they develop a pattern of high self-awareness and self-criticism, and negative self-evaluation. These aversive perceptions of themselves lead to emotional distress, including anxiety and low mood. They wish to escape these negative cognitive experiences and attempt to achieve this by engaging in binge eating. The escape theory suggests that the binge-eating episodes provide relief from these negative internal experiences by focusing the individual's attention on simple actions and sensations. Therefore, the escape theory suggests that the key to overcoming binge eating is to learn other methods of coping with these negative experiences (Paxton & Diggens, Reference Paxton and Diggens1997).

Blackburn, Johnston, Blampied, Popp, and Kallen (Reference Blackburn, Johnston, Blampied, Popp and Kallen2006) assessed the applicability of escape theory to binge eating using structural equation modelling (SEM) in a non-clinical sample. Their results provided support for this model as they found that the escape model provided a good fit to the data. They found that in their sample, perfectionism strongly predicted aversive self-awareness, which was found to strongly predict negative affect, which significantly predicted avoidant coping strategies, which in turn strongly predicted binge eating (Blackburn et al., Reference Blackburn, Johnston, Blampied, Popp and Kallen2006). Providing further support for the escape theory, Engelberg, Steiger, Gauvin, and Wonderlich (Reference Engelberg, Steiger, Gauvin and Wonderlich2007) examined the relationship between negative affect, dissociative experiences, and binge eating in a sample of women who met criteria for an eating disorder that involved regularly engaging in binge eating or purging (BN, BED, or an atypical eating disorder). The results of their study indicated that levels of negative affect and dissociation were significantly higher before a binge-eating episode compared to a meal or a snack (Engelberg et al., Reference Engelberg, Steiger, Gauvin and Wonderlich2007). Although the evidence supporting the escape theory as a stand-alone explanation of binge-eating pathology is somewhat limited, it shares many similarities with other theories related to binge eating as a mechanism for distress tolerance and emotional regulation.

Emotional Regulation Theory

Lacey (Reference Lacey, Downey and Malkin1986) also viewed binge eating as serving the function of reducing awareness of aversive emotional states but also proposed that binge eating might provide a way of coping with stressors. In his model, Lacey described predisposing factors as being adverse family circumstances, family history of weight and eating problems, a focus on physical appearance, and reliance on external standards to judge self-worth. Lacey proposed that these predisposing factors led to impairments in the individual's interpersonal functioning and their sense of identity, resulting in a sense of ineffectiveness and loss of control over their own body. Lacey identified that binge eating is triggered by a significant life event, often relating to loss, sexual conflict, or significant life changes that causes the individual to be self-critical or experience negative affect. Lacey asserted that these individuals lack alternative coping skills, and thus when they experience significant stress or distress they turn to food (bingeing or restricting intake) to moderate their emotional state. Binge eating is said to calm aroused emotional states such as anger or distress, or to provide distraction from boredom or loneliness (Lacey, Reference Lacey, Downey and Malkin1986). Thus, binge eating is maintained by the consequence of reducing negative affect (Lacey, Reference Lacey, Downey and Malkin1986).

Waller (Reference Waller1993) argued that focusing on specific symptoms, or symptom clusters, in eating disorders is a more clinically useful approach to understand the processes of eating disorders due to the large variability between individual cases presenting with the same diagnosis. As such, he focused on understanding binge eating as a symptom. McManus and Waller (Reference McManus and Waller1995) acknowledge the evidence that suggests that binge eating is triggered by food cravings (Grilo, Shiffman, & Carter-Campbell, Reference Grilo, Shiffman and Carter-Campbell1994) or dieting (Polivy & Herman, Reference Polivy and Herman1985), but they noted that this does not appear to be the case for all individuals who binge eat (Russell, Reference Russell1979). They presented the argument that previous leading cognitive theories of BN (such as those of Fairburn et al., Reference Fairburn, Cooper, Cooper, Brownell and Foreyt1986) focused on the role of dietary restraint as a crucial trigger. However, McManus and Waller argue that this does not provide a sufficient explanation of the phenomenon of binge eating as it does not account for the binge episodes experienced by those who have not restricted. They also argued that the restraint theory does not provide an adequate explanation of the motivation to binge eat, nor the benefits experienced by the individual (Beebe, Reference Beebe1994). McManus and Waller reviewed the evidence that looked at emotional consequences of binge eating. They cited evidence from clinical observation that reported that individuals experienced more stress, anxiety, and depression prior to the binge and that the negative affect is reduced during the binge, according to subjective reports (e.g., Chandarana & Malla, Reference Chandarana and Malla1989; Root & Fallon, Reference Root and Fallon1989). However, it has also been found that feelings of guilt, disgust and depression result after a binge episode, as well as anxiety about weight gain (McManus & Waller, Reference McManus and Waller1995). These feelings are then reduced by engaging in purging and compensatory behaviours that result in feelings of control and relief. However, following purging, the individual experiences depression and guilt. Thus, both bingeing and purging have been observed to reduce negative affect in the short term, but only insofar as they provide temporary relief or escape (McManus & Waller, Reference McManus and Waller1995).

There exists a substantial amount of experimental evidence supporting the emotional regulation model. For example, two independent laboratory experiments found that inducing a negative mood in women who had a diagnosis of BED resulted in a binge-eating episode and that a reduction in negative affect followed the binge episode (Agras & Telch, Reference Agras and Telch1998; Telch & Agras, Reference Telch and Agras1996). Indeed, a systematic review of 18 experimental studies examining the emotional regulation model in BED and obesity concluded that the results of the included studies supported the theory that negative emotion was an antecedent to binge eating for people with BED, and also that a short-term improvement in mood was experienced by participants following food intake in BED groups and also obesity (without BED) groups (Leehr et al., Reference Leehr, Krohmer, Schag, Dresler, Zipfel and Giel2015).

Results from naturalistic and observational studies with clinical samples also provide support for the emotional regulation model. For example, the antecedents of binge eating were examined in a naturalistic study of women who engaged in binge eating. Results indicated that self-reported levels of negative affect and dissociation were elevated prior to the commencement of binge-eating episodes (Engelberg et al., Reference Engelberg, Steiger, Gauvin and Wonderlich2007). Also, a study employing an ecological momentary assessment (EMA) method in a sample of individuals with a diagnosis of BED found that ratings for low mood were significantly higher (and ratings of positive mood significantly lower) on binge days compared to non-binge days (Munsch, Meyer, Quartier, & Wilhelm, Reference Munsch, Meyer, Quartier and Wilhelm2012). Munsch et al. (Reference Munsch, Meyer, Quartier and Wilhelm2012) also found that ratings of negative mood and tension increased significantly immediately prior to the binge-eating episode, and that subjective ratings of negative affect decreased and positive affect increased following a binge episode in people with BED. Munsch et al. concluded that these results indicated that binge eating in BED was triggered by a breakdown in emotional regulation attempts. In addition, studies have demonstrated a clear deficit in emotional regulation strategies in people with eating disorders compared to healthy controls (Lavender et al., Reference Lavender, Wonderlich, Engel, Gordon, Kaye and Mitchell2015). Although deficits in emotional regulation ability have been observed across different eating disorder diagnostic groups (AN, BN, BED) and other psychological conditions such as borderline personality disorder and major depressive disorder (Lavender et al., Reference Lavender, Wonderlich, Engel, Gordon, Kaye and Mitchell2015; Svaldi, Geirpenstroh, Tuschen-Caffier, & Ehring, Reference Svaldi, Griepenstroh, Tuschen-Caffier and Ehring2012), the results of Danner, Sternheim, and Evers (Reference Danner, Sternheim and Evers2014) demonstrate that the emotional regulation profile differs across the different eating disorder diagnostic groups. Interestingly, Danner et al. found that the emotional regulation profile of AN binge/purge subtype (AN-BP) was more similar to those of BN and BED than the AN restrictive type. However, it is important to note that some research has questioned the objectivity of the emotional regulation effect of binge eating; results from a meta-analysis of studies using EMA to assess negative affect before and after binge episodes did find that elevated levels of negative affect preceded binge-eating episodes; however, they did not find evidence that binge eating effectively reduced negative affect (Haedt-Matt & Keel, Reference Haedt-Matt and Keel2011). The authors of this meta-analysis note, however, that the studies included did not assess changes in affect during the binge-eating episodes and propose that it is possible that binge eating provides an immediate but time-limited, emotional regulation effect (Haedt-Matt & Keel, Reference Haedt-Matt and Keel2011).

Based on the emotional regulation model, dialectical behaviour therapy (DBT) has been used to treat binge eating because of its role in enhancing emotional regulation (Safer, Telch, & Agras, Reference Safer, Telch and Agras2001a, Reference Safer, Telch and Agras2001b). The efficacy of this type of treatment on reducing binge-eating frequency in individuals with BED (Telch, Agras, & Linehan, Reference Telch, Agras and Linehan2001) and BN (Safer et al., Reference Safer, Telch and Agras2001b) also provides evidence for the validity of the emotional regulation model of binge eating. Although, like CBT, DBT has been shown to provide short-term reductions in binge eating (89% of participants abstinent from binge eating at the end of the 20-week treatment; Telch et al., Reference Telch, Agras and Linehan2001), these gains are not entirely maintained (56% had remained abstinent at the 6-month follow-up; Telch et al., Reference Telch, Agras and Linehan2001), indicating that this treatment may also fail to address some important maintaining factors.

Building upon Lacey's (Reference Lacey, Downey and Malkin1986) formulation of binge eating, McManus and Waller (Reference McManus and Waller1995) provide a functional analysis of binge eating. They propose that people who binge eat have experienced particular predisposing factors that make that person more vulnerable to experiencing psychological difficulty. These predisposing factors include life dissatisfaction, a sense of ineffectiveness, low self-esteem (exacerbated by perfectionistic standards), a history of trauma, desire for control, and so on. They name two specific triggers of binge eating. The first trigger is food craving, believed to be resultant from dietary restriction. They account for the trigger of the dieting behaviour to be largely linked to the predisposing factors of need for control and societal pressures to be thin. The second trigger that they propose is the need to escape from aversive self-awareness and negative affect. This trigger is associated with many of the described predisposing factors such as low self-esteem and poor coping skills. They also described the consequences of binge eating as being an immediate reduction in negative affect and hunger, paired with longer term consequences of feelings of lack of control and anxiety about gaining weight, which serve to reinforce low self-esteem and increase effort to control their eating and weight further. In sum, McManus and Waller proposed that binge episodes are triggered by intolerable emotional states or hunger cues, that binge eating is maintained by the immediate effects of engaging in an episode — namely, reducing negative affect and food cravings — and that longer term effects, thought to exacerbate some of the original triggers, reinforce and maintain the cycle of behaviours.

Schema Theory of Binge Eating

Extending the theory of binge eating for emotional regulation to schema theory, Waller, Ohanian, Meyer, and Osman (Reference Waller, Ohanian, Meyer and Osman2000) examined the function of binge eating in alleviating the negative affect that occurs following a negative core belief being triggered. Core beliefs, or schema-level cognitions, are defined as absolute, unconditional and dichotomous negative cognitions about oneself, others and the world (J.E. Young, Reference Young1994). They are believed to develop early in life as a relatively adaptive response to the child's environment. But, over time and with changed circumstances, they become maladaptive. Examples of these unconditional beliefs or schemas include fear of abandonment and beliefs about being inadequate, defective, unlovable, or a failure. When one or more of these negative core beliefs is triggered, self-esteem is threatened and the individual may experience negative affect, such as low mood and anxiety, as a direct consequence.

Using the Young Schema Questionnaire (J.E. Young & Brown, Reference Young, Brown and Young1994), several studies have examined the core beliefs of individuals diagnosed with eating disorders (e.g., Leung, Thomas, & Waller, Reference Leung, Thomas and Waller2000; Waller, Meyer, & Ohanian, Reference Waller, Meyer and Ohanian2001; Waller et al., Reference Waller, Ohanian, Meyer and Osman2000). The results of these studies indicated that individuals with BED and BN experience significantly more pathological core beliefs than control participants. Certain schemas have been found to relate to particular eating disorder symptoms. Unoka, Tolgyes, Czobor, and Simon (Reference Unoka, Tölgyes, Czobor and Simon2010) investigated the presence of early maladaptive schemas in people with diagnosed eating disorders. Their study found that particular schemas (emotional deprivation, abandonment, enmeshment, subjugation, and emotional inhibition) are associated with eating disorder behaviours (binge eating and purging) and there are differences in schema profiles across the different diagnostic groups (AN, AN-BP, BN). Across multiple studies, binge eating has been found to be associated with a number of early maladaptive schemas; specifically, emotional inhibition, abandonment, vulnerability to harm, emotional deprivation, defectiveness/shame, failure to achieve, insufficient self-control, mistrust/abuse, social undesirability, and social isolation (Pugh, Reference Pugh2015). In particular, the ‘emotional inhibition’ schema has been found to be a good predictor of binge-eating frequency in BN, BED and AN-BP (Waller, Dickson & Ohanian, Reference Waller, Dickson and Ohanian2002; Waller et al., Reference Waller, Ohanian, Meyer and Osman2000; Waller et al., Reference Waller, Meyer and Ohanian2001). This schema relates to beliefs that emotions should not be experienced or expressed for fear of ridicule, shame, rejection, or feeling out of control. The schema model hypothesises that whenever the individual experiences strong feelings, they seek to inhibit their emotional response by either neutralising or avoiding their feelings. This is achieved by engaging in binge eating. In line with the theory of emotional regulation, episodes of binge eating function as a coping mechanism for dealing with what is perceived to be overwhelming negative affect (Waller et al., Reference Waller, Ohanian, Meyer and Osman2000).

Waller (Reference Waller2003) investigated the strength and type of core beliefs held by individuals with BED compared to BN or non-clinical controls, and then examined the relationship between the strength of certain core beliefs and the frequency of binge eating. The results of this study found that participants with either BN or BED demonstrated a higher level of pathological core beliefs than controls, but with a similar level to each other. This indicates that clinical participants experienced high levels of negative core beliefs, independent of their weight. Interestingly, BED participants endorsed different schemas on the Young Schema Questionnaire compared to the BN participants. Participants with a diagnosis of BED endorsed items pertaining to their inability to experience or express emotions, their inability to function independently, and their need to sacrifice their own needs for the needs for others more strongly than participants with BN, who endorsed items related to beliefs about abandonment more highly than those with BED. Another important finding was that the frequency of binge-eating episodes was associated with the strength of the core beliefs held, such that those who reported more episodes of binge eating were found to also have the highest levels of negative core beliefs. This particular finding was unique to the BED group; those in the BN group had high levels of negative cognitions regardless of the frequency of their binge episodes (Waller, Reference Waller2003). These results provide further evidence that there are important differences between BED and BN, and that it is not just the presence or absence of compensatory behaviours that separate the two diagnostic groups.

The evidence base for schema therapy in the treatment of binge eating is relatively limited, with only a few published studies examining the efficacy of schema-based therapy in this population (Masley, Gillanders, Simpson, & Taylor, Reference Masley, Gillanders, Simpson and Taylor2012; Pugh, Reference Pugh2015). A recent randomised controlled trial (RCT) assessed three different psychotherapies for the treatment of binge eating comparing schema therapy to CBT and an appetite-focused CBT treatment (McIntosh et al., Reference McIntosh, Jordan, Carter, Frampton, McKenzie, Latner and Joyce2016). The results of the study showed good outcomes for schema therapy in the treatment of binge eating, with significant reductions in binge frequency at the end of treatment; however, there were no significant differences in outcome between the three treatment modalities. McIntosh et al. (Reference McIntosh, Jordan, Carter, Frampton, McKenzie, Latner and Joyce2016) concluded that schema therapy and appetite-focused CBT provided suitable alternatives to CBT in the treatment of binge eating. Although schema therapy has some evidence indicating promising results, more research is needed in this area to determine the long-term efficacy of schema-based therapy in the treatment of binge eating.

Dual-Pathway Model

Stice and Agras (Reference Stice and Agras1998) proposed the dual-pathway model of bulimic pathology. This model also considers the influence of emotional regulation, as well as socio-cultural and dietary factors. The dual-pathway model asserts that the socio-cultural influences of the thin ideal are internalised by the individual and lead to body dissatisfaction. Pressures to be thin, which come from family, peers and/or the media, compound this. The dual-pathway model suggests that the body dissatisfaction experienced by the individual leads to dieting and negative affect — two factors that increase the risk of the individual engaging in a binge-eating episode. Not only is there a direct link between body dissatisfaction and negative affect, but the model also proposed that the act of dieting itself leads to increased negative affect due to both the failures associated with attempted weight loss and the effect of caloric deprivation on mood. Dieting is thought to lead to binge eating as either a physiological response to the caloric deprivation (as in the dietary restraint model) or because breaking a ‘strict rule’ of a diet can result in disinhibited eating (as in the abstinence violation effect). Thus, the dual-pathway model proposes that binge eating occurs as a response to either negative affect, dieting or a combination of the two (Stice & Agras, Reference Stice and Agras1998). Refer to Figure 2 for a representation of the dual-pathway model proposed by Stice and Agras.

Note: Reproduced from Stice (Reference Stice2001) with permission from the publisher.

FIGURE 2 Dual pathway model.

Stice (Reference Stice2001) tested the dual-pathway model using random regression growth curve models with a large community sample of adolescent women. Although the direct link between dieting and negative affect was found to be weak, the overall results of this analysis provided support for the dual-pathway model, supporting the model's hypotheses that pressure to be thin, thin-ideal internalisation, body dissatisfaction, dieting, and negative affect are risk factors for bulimia pathology (Stice, Reference Stice2001). A separate study examined the contributions of the proposed mediators of dieting (or dietary restraint) and negative affect in the relationship between body dissatisfaction and binge eating, as defined in the dual-pathway model (Holmes, Fuller-Tyszkiewicz, Skouteris, & Broadbent, Reference Holmes, Fuller-Tyszkiewicz, Skouteris and Broadbent2015). Holmes et al.’s (Reference Holmes, Fuller-Tyszkiewicz, Skouteris and Broadbent2015) results provide support for the dual-pathway model, finding that the mediators of dietary restraint and negative affect made significant unique contributions to the body dissatisfaction–binge eating relationship, accounting for 23.83% of the shared variance. Multiple studies examining the dual-pathway model have found support for a link between negative affect and binge eating (refer also to the section on the emotional regulation model); however, there is less consensus in the literature regarding the link between dietary restraint and binge eating. Both a longitudinal study (Stice, Shaw, & Nemeroff, Reference Stice, Shaw and Nemeroff1998) and a cross-sectional study (van Strien, Engels, van Leuwe, & Snoek, Reference Van Strien, Engels, Van Leeuwe and Snoek2005) examining the fit of the dual-pathway model in a clinical sample did not find a evidence of a significant causal relationship between dietary restraint and binge eating.

Further evidence supporting the dual pathway model for BN comes from prevention/intervention studies and prospective studies from Stice and colleagues. A dissonance-based eating disorder prevention program was developed based on the dual pathway model (refer to Stice, Chase, Stormer, & Appel, Reference Stice, Chase, Stormer and Appel2001). Long-term follow-up assessment of this prevention program provides strong evidence for the role of thin-ideal internalisation and body dissatisfaction in the development of eating disorder symptoms (Stice, Marti, Rohde, & Shaw, Reference Stice, Marti, Rohde and Shaw2011). Stice (Reference Stice2016) provides a review of multiple, large prospective studies and concludes that the risk factors of thin-ideal internalisation, perceived pressure to be thin, body dissatisfaction, dieting, and negative affect were found to be reliable predictors of later onset of BN. However, Stice (Reference Stice2016) did not identify any of the risk factors (or mediators) identified in the dual-pathway model to have predicted later onset of BED; the two studies identified that have investigated risk factors for BED (Stice et al., Reference Stice, Marti, Rohde and Shaw2011 and Stice et al., 2015, as cited in Stice, Reference Stice2016) found inconsistent results. Therefore, whether the dual-pathway model can be applied to BED remains unknown.

Transdiagnostic Cognitive-Behavioural Model of Eating Disorders

The important role of mood intolerance in maintaining binge eating pathology was acknowledged by Fairburn, Cooper, and Shafran (Reference Fairburn, Cooper and Shafran2003) in a revision of Fairburn's cognitive-behavioural model of BN. Fairburn et al. extended the focus of the model to incorporate additional maintaining mechanisms that had been found to interact with the original proposed core eating disorder mechanisms of weight, shape, and eating to create obstacles for change. In addition to acknowledging the role of mood intolerance, the maintaining mechanisms now included in the model concern the influence of clinical perfectionism, core low self-esteem and interpersonal difficulties (Fairburn et al., Reference Fairburn, Cooper and Shafran2003). In the same article, Fairburn et al. also proposed that the revised model for BN could function as a ‘transdiagnostic model’ for eating disorders, as they believe that all eating disorders are maintained by similar psychopathological processes. This transdiagnostic model embraces a broader range of maintaining mechanisms and can be used to formulate not only BN, but also AN, BED and atypical eating disorders. Refer to Figure 3 for a diagram of the transdiagostic model.

Note: Reproduced from Fairburn, Cooper, & Shafran (Reference Fairburn, Cooper and Shafran2003) with permission from the publisher.

FIGURE 3 Transdiagnostic model.

An independent study evaluated the utility of this enhanced transdiagnostic cognitive behavioural model compared to Fairburn's original model (Fairburn et al., Reference Fairburn, Cooper, Cooper, Brownell and Foreyt1986) with a BN sample (Lampard, Byrne, McLean, & Fursland, Reference Lampard, Byrne, McLean and Fursland2011). They found that both models provided a good fit to the data, but that the transdiagnostic model was able to account for a greater amount of variance in dietary restraint and binge eating than the original model. Similarly, Dalkanis et al. (Reference Dakanalis, Carrà, Calogero, Zanetti, Gaudio, Caccialanza and Clerici2015) used structural equation modelling to evaluate and compare these two models in a large clinical binge-eating sample. The results of this study found that both models provided a good fit to the data. However, the transdiagnostic model was found to provide greater explanatory power, accounting for a greater amount of variance in eating disorder symptoms than the original cognitive behavioural model. In a prospective study, three models of binge eating were evaluated and compared on their ability to fit the data (Allen, Byrne, & McLean, Reference Allen, Byrne and McLean2012). The results of this study showed that Fairburn's original cognitive behavioural model for BN (Fairburn, Reference Fairburn, Cooper, Cooper, Brownell and Foreyt1986) did not provide a good fit to the data, but that the trandiagnostic model (Fairburn et al., Reference Fairburn, Cooper and Shafran2003) and the dual-pathway model (Stice & Agras, Reference Stice and Agras1998) were more appropriate. However, the authors noted that although the fit statistics were acceptable, they were not excellent. They suggested that this result indicated that there may be additional variables involved that are not included in the transdiagnostic model or the dual-pathway model.

Based on the transdiagnostic model, Fairburn et al. (Reference Fairburn, Cooper, Shafran, Bohn, Hawker, Murphy, Straebler and Fairburn2008) developed an enhanced cognitive-behavioural transdiagnostic treatment program (CBT-E). Several clinical trials and RCTs have assessed the efficacy of CBT-E across different diagnostic groups. Fairburn et al. (Reference Fairburn, Cooper, Doll, O'Connor, Bohn, Hawker, Wales and Palmer2009) conducted a two-site trial of two versions of CBT-E, both involving 20 sessions of 50 minutes of individual psychotherapy. CBT-Ef was a ‘focused’ form of CBT-E in which session content is focused on pure eating disorder features (e.g., overconcern with shape and weight, dietary restriction, binge eating, and purging) and CBT-Eb which was a ‘broad’ form of CBT-E in which session content also addresses mood intolerance, perfectionism, poor self-esteem, and interpersonal functioning. The results of Fairburn et al.’s (Reference Fairburn, Cooper, Doll, O'Connor, Bohn, Hawker, Wales and Palmer2009) trial, and of a recent follow-up study by Fairburn et al. (Reference Fairburn, Bailey-Straebler, Basden, Doll, Jones, Murphy, O'Connor and Cooper2015) comparing CBT-Ef with interpersonal psychotherapy (IPT), are summarised in Table 1. Overall, a reduction in the number of participants reporting that they engaged in objective binge episodes (OBEs) occurs between pretreatment and post-treatment. This difference between pre- and post-OBEs is more pronounced for those participants receiving a CBT-E treatment than those receiving IPT. However, the differences between treatment groups in the number of participants reporting engaging in OBEs at the 60-week follow-up is less pronounced, with over a third of all patients still engaging in OBEs following treatment.

TABLE 1 Number of Participants Engaging in Objective Binge Eating Episodes (OBEs): Results From Two Papers Examining the Efficacy of CBT-E

Similar results were seen in an Australian-based open trial examining the efficacy of CBT-E for the treatment of eating disorders (Byrne, Fursland, Allen, & Watson, Reference Byrne, Fursland, Allen and Watson2011). This trial differed from Fairburn et al. (Reference Fairburn, Cooper, Doll, O'Connor, Bohn, Hawker, Wales and Palmer2009) as it included AN patients but did not include BED patients. Overall, 56.1% of patients were in full remission from their eating disorder at the end of treatment, with a further 10.6% in partial remission. For patients with BN, 50% of treatment completers were in full remission at the end of treatment, with a further 16.7% in partial remission at the end of treatment. There were significant differences between pre- and post-scores across a number of outcome measures, including improved quality of life and self-esteem, reduced depression, anxiety and stress scores, and a significant reduction in eating disorder psychopathology and symptoms. This included a significant reduction in the number of patients engaging in binge episodes between pre- and post-treatment. However, similar to the outcomes observed in Fairburn et al. (Reference Fairburn, Cooper, Doll, O'Connor, Bohn, Hawker, Wales and Palmer2009), out of 41 patients who were engaging in OBEs at pre-treatment, 15 (37%) were still engaging in OBEs at the end of treatment.

In addition, two recent studies examining the efficacy of 20 individual sessions of focused CBT-Ef with patients with BN revealed mixed results. In Poulsen et al. (Reference Poulsen, Lunn, Daniel, Folke, Mathiesen, Katznelson and Fairburn2014), 42% of patients had ceased binge eating and purging at the end of CBT-E treatment and these gains were maintained at the 5-month and 24-month follow-up. Wonderlich et al. (Reference Wonderlich, Peterson, Crosby, Smith, Klein, Mitchell and Crow2014) found a 76.3% reduction in OBEs at the end of CBT-E treatment. However, only 22.5% of participants were symptom free (i.e., abstinent from both binge eating and purging) at the end of treatment and at the 4-month follow-up. In both studies it was observed that a significant majority of participants (58% and 77.5%) were still engaging in bulimic behaviours following 20 sessions of one-on-one CBT-Ef psychotherapy.

Given the relatively low long-term efficacy of the best available treatment for binge eating, there have been a number of studies that have investigated possible predictors of outcome of CBT for BN and BED. For example, duration of illness, severity of eating disorder symptoms, presence of comorbid psychological disorders (e.g., personality disorders or depressive disorders), and higher frequency of binge eating at pretreatment have been found to predict worse outcomes for CBT-E treatment (Cooper et al., Reference Cooper, Allen, Bailey-Straebler, Basden, Murphy, O'Connor and Fairburn2016; Iacovino et al., Reference Iacovino, Gredysa, Altman and Wilfley2012). In the treatment of BN, increased dietary restraint at pretreatment has found to be predictive of better treatment outcomes, whereas higher depression scores at pretreatment were indicative of poorer treatment outcomes (Accurso et al., Reference Accurso, Wonderlich, Crosby, Smith, Klein, Mitchell and Peterson2016). In the treatment of BED, increased scores on drive for thinness and interoceptive awareness were found to predict better treatment outcomes, while binge-eating pathology and increased body dissatisfaction predicted poorer outcomes (Lammers, Vroling, Ouwens, Engels, & Strien, Reference Lammers, Vroling, Ouwens, Engels and Strien2015). In summary, when applied to a transdiagnostic sample, CBT-E demonstrates better long-term binge abstinence than its predecessor, CBT-BN. However, when the ‘focused’ form, CBT-Ef, is applied to a BN sample, over half of treated patients continue to engage in binge eating and purging behaviour at follow-up. The ‘broad’ version of CBT-Eb, with individualised components on mood tolerance, perfectionism, poor self-esteem and interpersonal functioning, seems to be the preferable form of CBT-E for treating binge eating. However, this outcome data still indicates that there may be components missing from the model that this treatment is based on that could be used to more effectively target, address, and eliminate binge-eating behaviour in patients.

Integrated Cognitive Behavioural Model of Eating Disorders

Williamson et al. (Reference Williamson, White, York-Crowe and Stewart2004) also proposed a transdiagnostic model that integrates a variety of hypotheses developed since the 1970s. This model provides a good summary of the overall theoretical understanding of eating disorders. The authors reviewed the literature and incorporated the main evidence-based hypotheses into their integrated cognitive behavioural theory of eating disorders. They included the factors of body self-schema, cognitive biases, negative emotional reaction, binge eating, compensatory behaviours, and risk factors for eating disorders. The results can be observed in Figure 4.

Note: Reproduced from Williamson, White, York-Crowe, and Stewart (Reference Williamson, White, York-Crowe and Stewart2004) with permission from the publisher.

FIGURE 4 Integrated cognitive behavioural model of eating disorders.

This model summarises the psychological risk factors for eating disorders as including a fear of fatness, overconcern with body shape and/or weight, and perfectionism. Central to this integrated theory is the self-schema related to body, weight, or eating. This has been emphasised by many theorists who have noted the overconcern with body shape and weight can lead to a body self-schema whereby the individuals will judge themselves based on internal and external cues related to their body shape and/or weight. This model hypothesises that people with this type of body-related self-schema will have their cognitive biases activated by certain stimuli such as body- or food-related information, ambiguous stimuli, and situations that require the individual to engage in self-reflection. The model proposes that the activation of the cognitive biases leads to negative emotions such as anxiety, feeling fat, depression, anger, and self-loathing. The individual experiences these negative emotions as aversive and feels compelled to escape or avoid these emotions. This is achieved by engaging in purging behaviours, restrictive eating, or excessive exercise. In this model, binge eating is seen both as a way to escape or avoid the aversive emotional experience, and also as a consequence of the dietary restriction. Following a binge episode, the individual engages in a number of eating disordered behaviours to reduce the distress caused by binge eating and/or activation of the cognitive biases. These behaviours effectively reduce distress, which negatively reinforces the behaviours, confirms the individual's reliance on these avoidance behaviours, and serves to confirm their fear of fatness, overconcern with body shape and/or weight, internalisation of a thin ideal, and need for perfectionism or obessionality. Williamson et al. (Reference Williamson, White, York-Crowe and Stewart2004) propose that this in turn serves to reinforce the body-related self-schema.

The integrated model provides a good summary of much of the leading literature on this topic. It considers the theoretical contributions of dietary restraint theory, emotional regulation theory, the dual pathway model, schema theory, and core concepts that were proposed in Fairburn's cognitive behavioural model. The evidence for this model comes from the evidence that supports all the models that they have integrated (Williamson et al., Reference Williamson, White, York-Crowe and Stewart2004). This model has not been directly tested for its fit to the data, nor has there been any published treatment methods that are based on this integrated model. While this model encapsulates many processes that have been identified as contributing to eating disorder behaviour, none of the models summarised here so far have investigated whether individuals who binge eat have specific beliefs about food that may function to maintain binge-eating behaviour.

Cognitive Model of BN

M.J. Cooper, Wells, and Todd (Reference Cooper, Wells and Todd2004) proposed a model for BN with the aim of addressing some shortfalls in the cognitive models proposed thus far. They based their model on the results of detailed semi-structured interviews with individuals meeting diagnostic criteria for BN. In their interviews they collected information about the thoughts and feelings experienced by the individuals before, during, and after their most recent binge episode, as well as information regarding their beliefs, their early experiences, and their opinions on compensatory behaviours and dieting (data from these interviews are presented in M. Cooper, Todd, & Wells, Reference Cooper, Todd and Wells1998). M.J. Cooper et al.’s (Reference Cooper, Wells and Todd2004) model also drew heavily on much of the research summarised above, including the work of Bruch (Reference Bruch and Vigersky1977), the escape theory of Heatherton and Baumeister (Reference Heatherton and Baumeister1991), and theories that describe binge eating as serving the function of coping with negative thoughts and feelings, such as in emotional regulation theory. M.J. Cooper et al. also drew inspiration from leading contemporary cognitive theories such as Beck's general schema theory (Beck, Freeman, & Davis, Reference Beck, Freeman and Davis2006), J.E. Young's (Reference Young1994) work on schema-driven processes, Padesky and Greenberger's (Reference Padesky and Greenberger1995) work on the difference between core beliefs and underlying assumptions, and work by Wells (Reference Wells1993), which suggested that some disorders are maintained by conflicting and interacting beliefs about symptoms and functions of behaviours and unhelpful coping strategies.

The model proposed by M.J. Cooper et al. (Reference Cooper, Wells and Todd2004) focuses on maintenance processes and developmental factors. The maintenance processes are presented as a cycle of behaviours that are driven by core beliefs and related meta-cognitive beliefs. M.J. Cooper et al. theorise that a binge-eating episode is triggered by a distressing event that activates a negative belief about the self as an acceptable person, such as ‘I'm unlovable’ or ‘I'm a failure’. The activation of these negative self-appraisals is accompanied by feelings of anxiety, depression and/or guilt. The model suggests that these individuals engage in binge eating in order to cope with these emotions, and the process of eating reduces the intensity of the emotional states, which reinforces positive beliefs about eating (e.g., ‘Eating helps me to feel better’). M.J. Cooper et al. describe a conflict experienced by those with BN as having both positive and negative beliefs about eating. This conflict is distressing, and so M.J. Cooper et al. argue that additional meta-cognitive beliefs about eating develop to reduce this distress. These beliefs are categorised as permissive beliefs (e.g., ‘It's okay to binge’) and beliefs related to loss of control (e.g., ‘I cannot control my eating’). They suggest that it is the activation of permissive thoughts and thoughts of ‘no control’ that lead to a binge episode. The cycle continues as binge eating activates negative self-beliefs and negative thoughts about eating (e.g., ‘I'll get fat’) that lead to the behaviour of purging, which then serves to alleviate some of the distress caused by the binge-eating episode. M.J. Cooper et al. also describe a role for bodily sensations such as hunger in triggering a binge episode. They suggest that when hunger is a triggering factor, it activates thoughts of no control, which precipitates binge eating. Figure 5 presents a representation of this model.

Note: Reproduced from M.J. Cooper, Wells, and Todd (Reference Cooper, Wells and Todd2004) with permission from the publisher.

FIGURE 5 Cognitive model of bulimia nervosa.

M.J. Cooper et al. (Reference Cooper, Wells and Todd2004) described the developmental factors that they believe contribute to the development of BN. They identified that negative or traumatic experiences in early childhood or adolescence were found to lead to the development of dysfunctional beliefs, such as negative self-beliefs. Dieting is a method used by these individuals to cope with their negative self-beliefs; this is driven by the underlying assumptions that dieting will lead to acceptance by others or will allow them to accept themselves, as well as the development of negative beliefs about the consequences of gaining weight, which also serve to further reinforce the dieting behaviour.

M.J. Cooper et al.’s (Reference Cooper, Wells and Todd2004) model is unique in the way in which it describes the role of positive, negative, and permissive metacognitive beliefs about eating in maintaining binge eating. M.J. Cooper, Todd, Woolrich, Somerville, and Wells (Reference Cooper, Todd, Woolrich, Somerville and Wells2006) developed a measure to assess the maintaining metacognitive beliefs proposed in Cooper's cognitive model of BN (M. Cooper et al., Reference Cooper, Wells and Todd2004), the Eating Disorder Thoughts Questionnaire (EDTQ). This measure contained items that provided examples of positive, negative, and permissive beliefs about eating. The results of the discriminant validity analysis for this questionnaire found that the EDTQ could discriminate between people with a diagnosis of AN, BN, dieters, and controls. A different questionnaire was developed that aimed to assess the core negative self-beliefs hypothesised to maintain BN in M.J. Cooper et al.’s (Reference Cooper, Wells and Todd2004) model, the Eating Disorder Core Beliefs Questionnaire (ED-CBQ; Fairchild & Cooper, Reference Fairchild and Cooper2010). In their preliminary findings on the validity of the ED-CBQ, Fairchild and Cooper (Reference Fairchild and Cooper2010) found that items pertaining to self-loathing were significantly associated with eating disorder symptoms, both AN and BN. Young and Cooper (Reference Young and Cooper2013) investigated whether M.J. Cooper's cognitive model for bulimia could be applied to binge eating in a community sample. They assessed the presence of positive, negative, and permissive beliefs using the EDTQ (M.J. Cooper et al., Reference Cooper, Todd, Woolrich, Somerville and Wells2006) and negative self-beliefs using the ED-CBQ (Fairchild & Cooper, Reference Fairchild and Cooper2010). Their results indicated that these three categories of eating disorder thoughts (negative, positive, and permissive), as well as two negative beliefs (self-loathing and demanding/needing help and support) significantly predicted variance in binge-eating pathology in a community sample. Bergin and Wade (Reference Bergin and Wade2012) tested the M.J. Cooper et al. model with a sample of individuals with BN and control participants. Consistent with the model, they found that negative self-beliefs were associated with negative affect, that negative affect was associated with both positive and negative thoughts about eating, that beliefs about eating were associated with eating behaviours, and that binge eating was associated with negative self-beliefs. They also found some inconsistencies with the model: they did not find an association between compensatory behaviour and negative self-beliefs, nor did they find any significant association of any behaviours with permissive thoughts. The authors noted that using a predominantly non-clinical sample may have limited the results of this study and they recommended more research was required to further develop and assess the model.

Based on this model, M. Cooper, Todd, and Wells (Reference Cooper, Todd and Wells2000) developed a cognitive therapy self-help treatment manual for BN that could be used with or without the assistance of a therapist (i.e., guided self-help). Pritchard, Bergin, and Wade (Reference Pritchard, Bergin and Wade2004) provided a preliminary evaluation of this treatment approach using a case series of 15 patients with BN who attended six 30-minute guided self-help (GSH) sessions with clinicians. Pritchard et al. found significant improvements in eating disorder symptoms and attitudes between pretreatment and post-treatment scores. This included binge eating, which was reduced by 73% at post-treatment, and by 74% at the 3-month follow-up assessment. However, a third of patients still met clinical criteria for BN at the end of treatment and at the 3-month follow-up. Though such a significant reduction in binge-eating epsiodes is a promising result from a GSH program, it is likely that for this population, a more intensive, clinician-direction treatment approach would result in improved long-term outcomes.

M. Cooper, Todd, and Wells (Reference Cooper, Todd and Wells2008) also developed a treatment manual for clinicians based on their cognitive model for BN (M.J. Cooper et al., Reference Cooper, Wells and Todd2004). They published preliminary findings of the results of an A-B single-case replication series (M. Cooper, Todd, Turner, & Wells, Reference Cooper, Todd, Turner and Wells2007). Their study used a sample of three patients. All patients no longer met DSM-IV criteria for BN at the end of the 14–20 sessions of treatment. Two of the three were completely symptom free at the 6-month follow-up, and the third was symptom free at the 9-month follow-up. The findings of both Pritchard et al. (Reference Pritchard, Bergin and Wade2004) and M. Cooper et al. (Reference Cooper, Todd, Turner and Wells2007) are promising, but with significant limitations such as small sample sizes and no comparison groups. In order to thoroughly examine the efficacy of this treatment, a randomised controlled trial with a larger sample size should be undertaken.

Despite some promising preliminary results, empirical evidence supporting M.J. Cooper et al.’s (Reference Cooper, Wells and Todd2004) theoretical understanding of binge eating is left wanting. Although this model is believed to provide a formulation of BED as well as BN, this model has not yet been validated with a clinical binge-eating group, nor with a group of individuals with a diagnosis of BED. In order to establish whether the model presented by M.J. Cooper et al. provides a sound and evidence-based model of binge eating in BN or BED, it is important that the model is tested with a clinical sample. It would be useful for future research to compare fit statistics of the different leading models that are currently used to formulate binge-eating pathology such as Fairburn's transdiagnostic model (Fairburn et al., Reference Fairburn, Cooper and Shafran2003), Williamson's integrated model (Williamson et al., Reference Williamson, White, York-Crowe and Stewart2004), and Cooper's cognitive model of BN (M.J. Cooper et al., Reference Cooper, Wells and Todd2004).