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Chapter 5 - Expanding the concept of dementia

Published online by Cambridge University Press:  05 May 2016

Christopher M. Filley
Affiliation:
University of Colorado School of Medicine
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Publisher: Cambridge University Press
Print publication year: 2016

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References

Albert, ML. Subcortical dementia. In: Katzman, R, Terry, RD, Bick, KL, eds. Alzheimer’s disease: senile dementia and related disorders. New York: Raven Press, 1978: 173180.Google Scholar
Albert, ML. Subcortical dementia: historical review and personal view. Neurocase 2005; 11: 243245.CrossRefGoogle ScholarPubMed
Appelman, AP, Exalto, LG, van der Graaf, Y, et al. White matter lesions and brain atrophy: more than shared risk factors? A systematic review. Cerebrovasc Dis. 2009; 28: 227242.CrossRefGoogle ScholarPubMed
Bangert-Drowns, RL. Misunderstanding meta-analysis. Eval Health Prof 1995; 18: 304314.CrossRefGoogle ScholarPubMed
Bartzokis, G. Brain myelination in prevalent neuropsychiatric developmental disorders: primary and comorbid addiction. Adolesc Psychiatry 2005; 29: 5596.Google ScholarPubMed
Bartzokis, G. Alzheimer’s disease as homeostatic responses to age-related myelin breakdown. Neurobiol Aging 2011; 32: 13411371.CrossRefGoogle ScholarPubMed
Bonelli, RM, Cummings, JL. Frontal-subcortical dementias. Neurologist 2008; 14: 100107.CrossRefGoogle ScholarPubMed
Casadevall, A, Fang, FC. Descriptive science. Infect Immun 2008; 76: 38353836.CrossRefGoogle ScholarPubMed
Charcot, JM. Lectures on diseases of the nervous system delivered at La Salpêtrière. London: New Sydenham Society, 1877.Google Scholar
Cook, DJ, Mulrow, CD, Haynes, RB. Systematic reviews: synthesis of best evidence for clinical decisions. Ann Intern Med 1997; 126: 376380.CrossRefGoogle ScholarPubMed
Cummings, JL, Benson, DF. Dementia: a clinical approach. Boston: Butterworths, 1983.Google Scholar
Cummings, JL, Benson, DF. Subcortical dementia: review of an emerging concept. Arch Neurol 1984; 41: 874879.CrossRefGoogle ScholarPubMed
Debette, S, Markus, HS. The clinical importance of white matter hyperintensities on brain magnetic resonance imaging: systematic review and meta-analysis. BMJ 2010; 341: c3666.CrossRefGoogle ScholarPubMed
DeKosky, ST. The role of big data in understanding late-life cognitive decline. JAMA Neurology 2014; 71: 14761478.CrossRefGoogle ScholarPubMed
Fang, FC, Casadevall, A. Reductionistic and holistic science. Infect Immun 2011; 79: 14011404.CrossRefGoogle ScholarPubMed
Fields, RD. White matter in learning, cognition and psychiatric disorders. Trends Neurosci 2008; 31: 361370.CrossRefGoogle ScholarPubMed
Fields, RD. Neuroscience. Myelin – more than insulation. Science 2014; 344: 264266.CrossRefGoogle ScholarPubMed
Filley, CM. Neurobehavioral aspects of cerebral white matter disorders. Psychiatr Clin North Am 2005; 28: 685700.CrossRefGoogle ScholarPubMed
Filley, CM. White matter: beyond focal disconnection. Neurol Clin 2011; 29: 8197.CrossRefGoogle ScholarPubMed
Filley, CM. The behavioral neurology of white matter. 2nd ed. New York: Oxford University Press, 2012.CrossRefGoogle ScholarPubMed
Filley, CM, Rollins, YD, Anderson, CA, et al. The genetics of very early onset Alzheimer’s Disease. Cogn Behav Neurol 2007; 20: 149156.CrossRefGoogle Scholar
Gray, RG, Preece, MA, Green, SH, et al. Inborn errors of metabolism as a cause of neurological disease in adults: an approach to investigation. J Neurol Neurosurg Psychiatry 2000; 69: 512.CrossRefGoogle ScholarPubMed
Grimes, DA, Schulz, KF. Descriptive studies: what they can and cannot do. Lancet 2002; 359: 145149.CrossRefGoogle Scholar
Haroutunian, V, Katsel, P, Roussos, P, et al. Myelination, oligodendrocytes, and serious mental illness. Glia 2014; 62: 18561877.CrossRefGoogle ScholarPubMed
Inatomi, Y, Yonehara, T, Hashimoto, Y, et al. Correlation between ventricular enlargement and white matter changes. J Neurol Sci 2008; 269: 1217.CrossRefGoogle ScholarPubMed
Inzitari, D, Pracucci, G, Poggesi, A, et al. Changes in white matter as determinant of global functional decline in older independent outpatients: three year follow-up of LADIS (leukoaraiosis and disability) study cohort. BMJ 2009; 339: b2477.CrossRefGoogle ScholarPubMed
Kelley, BJ, Boeve, BF, Josephs, KA. Young-onset dementia: demographic and etiologic characteristics of 235 patients. Arch Neurol 2008; 65: 15021508.CrossRefGoogle ScholarPubMed
Kozora, E, Filley, CM. Cognitive dysfunction and white matter abnormalities in systemic lupus erythematosus. J Int Neuropsychol Soc 2011; 22: 18.Google Scholar
Kuruppu, DK, Matthews, BR. Young-onset dementia. Semin Neurol 2013; 33: 365385.CrossRefGoogle ScholarPubMed
Lafosse, JM, Corboy, JR, Leehey, MA, et al. MS vs. HD: Can white matter and subcortical gray matter pathology be distinguished neuropsychologically? J Clin Exp Neuropsychol 2007; 29: 142154.CrossRefGoogle ScholarPubMed
McKee, AC, Stein, TD, Nowinski, CJ, et al. The spectrum of disease in chronic traumatic encephalopathy. Brain 2013; 136: 4364.CrossRefGoogle ScholarPubMed
Mendez, MF. The accurate diagnosis of early-onset dementia. Int J Psychiatry Med 2006; 36: 401412.CrossRefGoogle ScholarPubMed
Mighdoll, MI, Tao, R, Kleinman, JE, Hyde, TM. Myelin, myelin-related disorders, and psychosis. Schizophr Res 2015; 161: 8593.CrossRefGoogle ScholarPubMed
Parvizi, J. Corticocentric myopia: old bias in new cognitive sciences. Trends Cogn Sci 2009; 13: 354359.CrossRefGoogle ScholarPubMed
Peterson, RL, Pennington, BF. Developmental dyslexia. Lancet 2012; 379: 19972007.CrossRefGoogle ScholarPubMed
Rossor, MN, Fox, NC, Mummery, CJ, et al. The diagnosis of young-onset dementia. Lancet Neurol 2010; 9: 793806.CrossRefGoogle ScholarPubMed
Thomas, MS, Knowland, VC, Karmiloff-Smith, A. Mechanisms of developmental regression in autism and the broader phenotype: a neural network modeling approach. Psychol Rev 2011; 118: 637654.CrossRefGoogle ScholarPubMed
Tuladhar, AM, Reid, AT, Shumskaya, E, et al. Relationship between white matter hyperintensities, cortical thickness, and cognition. Stroke 2015; 46: 425432.CrossRefGoogle ScholarPubMed
Walterfang, M, Wood, SJ, Velakoulis, D, et al. Diseases of white matter and schizophrenia-like psychosis. Aust N Z J Psychiatry 2005; 39: 746756.CrossRefGoogle ScholarPubMed
Whittemore, R, Chao, A, Jang, M, et al. Methods for knowledge synthesis: an overview. Heart Lung 2014; 43: 453461.CrossRefGoogle ScholarPubMed

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