1. , , , and (2007). The contribution of gene–environment interaction to psychopathology. Development and Psychopathology, 19, 989–1004.
2. and (2007). Genetic expression outside the skin: Clues to mechanisms of genotype × environment interaction. Development and Psychopathology, 19, 1005–1027.
3. . (2008). Commentary on “The case for gene–environment interactions in psychiatry.” Current Opinion in Psychiatry, 21, 326–327.
4. , , et al. (2008). Forum: The interplay of genes and environment in psychiatric disorders. Current Opinion in Psychiatry, 21, 322–323.
5. and . (2008). Forum: Interactions between gene and environment. Current Opinion in Psychiatry, 21, 315–317.
6. . (2008). Biological Implications of gene–environment Interaction. Journal of Abnormal Child Psychology, 36, 969–975.
7. , and (2006). Measured gene–environment interactions in psychopathology. Perspectives on Psychological Science, 1, 5–27.
8. and (2007). The Neurobiology of Stress and Development. Annual Reviews in Psychology, 58, 145–173.
9. , and . (2007). Plasticity for affective neurocircuitry: How the environment affects gene expression. Current Directions in Psychological Science, 16, 1–5.
10. , , and (2008). Gene × environment interactions at the serotonin transporter locus. Biological Psychiatry, 13, 131–146.
11. and (2008). Depression and the serotonin transporter 5-HTTLPR polymorphism: A review and a hypothesis concerning gene–environment interaction. Journal of Affective Disorders, 111, 1–12.
12. and (2007). Meta-analysis of gene–environment interactions in developmental psychopathology. Development and Psychopathology, 19, 1029–1037.
13. , , et al. (1995). Stressful life events, genetic liability, and onset of an episode of major depression in women. American Journal of Psychiatry, 152, 833–842.
14. , et al. (1996). Association of anxiety-related traits with a polymorphism in the serotonin transporter gene regulatory region. Science, 274, 1527–1531.
15. , , et al. (2003). Influence of life stress on depression: Moderation by a polymorphism in the 5-HTT gene. Science, 301, 386–389.
16. (2006). Risk, resilience, and gene × environment Interactions in Rhesus monkeys. Annals of the New York Academy of Sciences, 1094, 52–62.
17. , , et al. (2006). Early family environment, current adversity, the serotonin transporter promoter polymorphism, and depressive symptomatology. Biological Psychiatry, 60, 671–676.
18. , , , and (2005). The interaction of stressful life events and a serotonin transporter polymorphism in the prediction of episodes of major depression: A replication. Archives of General Psychiatry, 62, 529–535.
19. , , and (2005). The relationship between stressful life events, the serotonin transporter (5-HTTLPR) genotype, and major depression. Psychological Medicine, 35, 101–111.
20. , , et al. (2006). Social adversity, the serotonin transporter (5-HTTLPR) polymorphism and major depressive disorder. Biological Psychiatry, 59, 224–229.
21. , et al. (2004). Gene–environment interaction analysis of serotonin system markers with adolescent depression. Molecular Psychiatry, 9, 908–915.
22. , et al. (2005). Mental and physical distress is modulated by a polymorphism in the 5-HT transporter gene interacting with social stressors and chronic disease burden. Molecular Psychiatry, 10, 220–224.
23. , and (2007). Gene-by-environment (serotonin transporter and childhood maltreatment) interaction for anxiety sensitivity, an intermediate phenotype for anxiety disorders. Neuropsychopharmacology, 33, 312–319.
24. , , et al. (2007). Serotonin transporter gene polymorphism (5-HTTLPR) and anxiety reactivity in daily life: A daily process approach to gene–environment interaction. Psychosomatic Medicine, 69, 762–768.
25. , et al. (2006). Brain-derived neurotrophic factor-5-HTTLPR gene interactions and environmental modifiers of depression in children. Biological Psychiatry, 59, 673–680.
26. , et al. (2004). Social supports and serotonin transporter gene moderate depression in maltreated children. Proceedings of the National Academy of Sciences of the United States of America, 101, 17316–17321.
27. and (2005). The neurobiology of depression: Inroads to treatment and new drug discovery. Journal of Clinical Psychiatry, Supplement, 66, 5–13.
28. , , et al. (2005). Long-term consequences of neonatal rearing on central corticotropin-releasing factor systems in adult male rat offspring. Neuropsychopharmacology, 30, 2192–2204.
29. , et al. (2000). Pituitary–adrenal and autonomic responses to stress in women after sexual and physical abuse in childhood. Journal of the American Medical Association, 284, 592–597.
30. , et al. (2007). Convergent genetic modulation of the endocrine stress response involves polymorphic variations of 5-HTT, COMT and MAOA. Molecular Psychiatry, 12, 483–490.
31. , and (2007). HPA axis reactivity: A mechanism underlying the associations among 5-HTTLPR, stress, and depression. Biological Psychiatry, 9, 847–851.
32. et al. (2004). Epigenetic programming by maternal behavior. Nature Neuroscience, 7, 847–854.
33. , , et al. (2008). Influence of child abuse on adult depression: Moderation by the corticotropin-releasing hormone gene. Archives of General Psychiatry, 65, 190–200.
34. , , et al. (2000). Long-term behavioral and neuroendocrine adaptations to adverse early experience. Progress in Brain Research, 122, 81–103.
35. et al. (2008). Association of FKBP5 polymorphisms and childhood abuse with risk of posttraumatic stress disorder symptoms in adults. Journal of the American Medical Association, 299, 1291–1305.
36. , et al. (2004). Polymorphisms in FKBP5 are associated with increased recurrence of depressive episodes and rapid response to antidepressant treatment. Nature Genetics, 36, 1319–1325.
37. , , et al. (2005). 5-HTTLPR polymorphism impacts human cingulate–amygdala interactions: A genetic susceptibility mechanism for depression. Nature Neuroscience, 8, 828–834.
38. et al. (2002). Role of genotype in the cycle of violence in maltreated children. Science, 297, 851–854.
39. et al. (2006). MAOA, maltreatment, and gene–environment interaction predicting children’s mental health: New evidence and a meta-analysis. Molecular Psychiatry, 11, 903–913.
40. , , et al. (2004). Childhood adversity, monoamine oxidase a genotype, and risk for conduct disorder. Archives of General Psychiatry, 61, 738–744.
41. , , et al. (2004). An association between a functional polymorphism in the monoamine oxidase A gene promoter, impulsive traits and early abuse experiences. Neuropsychopharmacology,29, 1498–1505.
42. , et al. (2006). Childhood maltreatment, subsequent antisocial behavior, and the role of monoamine oxidase a genotype. Biological Psychiatry, 60, 677–683.
43. , , et al. (2006). Neural mechanisms of genetic risk for impulsivity and violence in humans. Proceedings of the National Academy of Sciences United States of America, 103, 6269–6274.
44. , , and (2003). Role of dopamine transporter genotype and maternal prenatal smoking in childhood hyperactive–impulsive, inattentive, and oppositional behaviors. Journal of Pediatrics, 143, 104–110.
45. , , et al. (2007). Interacting effects of the dopamine transporter gene and psychosocial adversity on attention-deficit/hyperactivity disorder symptoms among 15-year-olds from a high-risk community sample. Archives of General Psychiatry, 64, 585–590.
46. , , and (2007). Parenting quality interacts with genetic variation in dopamine receptor D4 to influence temperament in early childhood. Development and Psychopathology, 19, 1039–1046.
47. and (2006). Gene–environment interaction of the dopamine D4 receptor (DRD4) and observed maternal insensitivity predicting externalizing behavior in preschoolers. Developmental Psychobiology, 48, 406–409.
48. , and (2008). Dopamine system genes associated with parenting in the context of daily hassles. Genes, Brain, and Behavior, 7, 403–410.
49. , and (2007). For better and for worse: Differential susceptibility to environmental influences. Current Directions in Psychological Science, 16, 300–304.
50. , , , and (2008). Effects of an attachment-based intervention on daily cortisol moderated by dopamine receptor D4: A randomized control trial on 1- to 3-year-olds screened for externalizing behavior. Development and Psychopathology, 20, 805–820.
51. , and et al. (2006). The enduring effects of abuse and related adverse experiences in childhood. A convergence of evidence from neurobiology and epidemiology. European Archives of Psychiatry and Clinical Neuroscience, 256, 174–186.
52. and (2008). Stress, sensitive periods and maturational events in adolescent depression. Trends in Neurosciences, 31, 183–191.
53. (2007). The developmental consequences of child emotional abuse: A neurodevelopmental perspective. Journal of Emotional Abuse, 7, 9–34.