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Dietary antioxidant indices (DAI) may be potentially associated with relative telomere length (RTL) of leucocytes. This study aimed to investigate the relationship between DAI and RTL. A cross-sectional study involving 1656 participants was conducted. A generalised linear regression model and a restricted cubic spline model were used to assess the correlation of DAI and its components with RTL. Generalised linear regression analysis revealed that DAI (β = 0·005, P = 0·002) and the intake of its constituents vitamin C (β = 0·043, P = 0·027), vitamin E (β = 0·088, P < 0·001), Se (β = 0·075, P = 0·003), and Zn (β = 0·075, P = 0·023) were significantly and positively correlated with RTL. Sex-stratified analysis showed that DAI (β = 0·006, P = 0·005) and its constituents vitamin E (β = 0·083, P = 0·012), Se (β = 0·093, P = 0·006), and Zn (β = 0·092, P = 0·034) were significantly and positively correlated with RTL among females. Meanwhile, among males, only vitamin E intake (β = 0·089, P = 0·013) was significantly and positively associated with RTL. Restricted cubic spline analysis revealed linear positive associations between DAI and its constituents’ (vitamin E, Se and Zn) intake and RTL in the total population. Sex-stratified analysis revealed a linear positive correlation between DAI and its constituents’ (vitamin E, Se and Zn) intake and RTL in females. Our study found a significant positive correlation between DAI and RTL, with sex differences.
The science of histopathology revealed that like normal tissues, tumors are composed of cells. During development and in physiological contexts, proliferation is regulated by exposure of cells to soluble growth factors within their environment. Cells use a number of distinct signaling pathways to control their proliferation. A cell's decision to divide or to become quiescent is influenced by mitogenic signals in the cell's surroundings. In rapidly dividing tissues, parenchymal cells are born from asymmetric division of a stem/progenitor cell, differentiate and then undergo programmed cell death. Intact DNA damage repair systems are critical to maintain genomic stability and prevent tumorigenesis. Inactivation of the apoptotic machinery permits the survival of cells with accumulatingmutations and promotes evolution of premalignant to malignant cells. Failure in the ability of the immune system to distinguish self from non-self can result in autoimmune reactions or facilitate the development of a tumor.
The degradation mechanism of CdSe/ZnS quantum dots (QDs) light-emitting
diodes (LEDs) was investigated with steady-state and time-resolved
photoluminescence measurements. Our study reveals that the degradation is
associated with the decreasing quantum efficiency of the CdSe/ZnS QDs in the
devices. Two mechanisms that cause the efficiency reduction were verified in
the experiments: i.e., thermal instability and luminescence quenching.
We presented in this paper a photo-assisted ligand exchange approach whereby
light will be introduced to facilitate the replacement of oleic acid (OA)
ligand molecules over PbSe quantum dots (QDs). The ligand-exchanged QDs were
used to fabricate quantum dot light-emitting-diodes (QD-LEDs), which
outperform the devices comprising the QDs without ligand-replacement.
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