To save content items to your account,
please confirm that you agree to abide by our usage policies.
If this is the first time you use this feature, you will be asked to authorise Cambridge Core to connect with your account.
Find out more about saving content to .
To save content items to your Kindle, first ensure email@example.com
is added to your Approved Personal Document E-mail List under your Personal Document Settings
on the Manage Your Content and Devices page of your Amazon account. Then enter the ‘name’ part
of your Kindle email address below.
Find out more about saving to your Kindle.
Note you can select to save to either the @free.kindle.com or @kindle.com variations.
‘@free.kindle.com’ emails are free but can only be saved to your device when it is connected to wi-fi.
‘@kindle.com’ emails can be delivered even when you are not connected to wi-fi, but note that service fees apply.
This chapter discusses the mechanisms of spinal cord plasticity in animal models as revealed by the recovery of motor functions after a spinal lesion. It shows that in cats, rats and mice, motor programs such as locomotion are re-expressed after a complete spinal transection at the low-thoracic level. The pharmacological work with intrathecal cannula suggested that important effects on locomotion could be observed when the intrathecal injections were localized to the rostral spinal segments. Clinically, the notion that certain spinal segments may play a critical role in the control of spinal locomotion may help to somewhat simplify where to target pharmacological stimulation, cell grafts or electrical stimulation. With the advent of genetic characterization and the potential for genetic manipulations, the mouse is becoming an increasingly important model for spinal cord injury research. The characteristics of motor patterns result from an intricate dynamic sensori-motor interaction between the spinal and supraspinal levels.
Email your librarian or administrator to recommend adding this to your organisation's collection.