To save content items to your account,
please confirm that you agree to abide by our usage policies.
If this is the first time you use this feature, you will be asked to authorise Cambridge Core to connect with your account.
Find out more about saving content to .
To save content items to your Kindle, first ensure firstname.lastname@example.org
is added to your Approved Personal Document E-mail List under your Personal Document Settings
on the Manage Your Content and Devices page of your Amazon account. Then enter the ‘name’ part
of your Kindle email address below.
Find out more about saving to your Kindle.
Note you can select to save to either the @free.kindle.com or @kindle.com variations.
‘@free.kindle.com’ emails are free but can only be saved to your device when it is connected to wi-fi.
‘@kindle.com’ emails can be delivered even when you are not connected to wi-fi, but note that service fees apply.
This chapter focuses on advances towards heritability and genetic approaches in insomnia disorders in human. Insomnia remains a heterogeneous condition that is primarily characterized and diagnosed by subjective complaints about dissatisfaction with sleep quantity or quality, and it is not associated with any specific biomarker. Some of the first twin studies with a focus on insomnia suggest the involvement of genetic factors in early-onset (childhood) insomnia. The first familial study on insomnia using a clinic based sample suggests the presence of familial insomnia aggregations, especially among individuals with childhood or adolescence onset compared to those with adult onset. Twin studies strongly suggest that genetic factors may trigger insomnia with genetic effects accounting for approximately one-third of the variance in insomnia complaints. Results from twin studies suggest that heritability would potentially account for large proportion of variance in insomniac symptoms.
Email your librarian or administrator to recommend adding this to your organisation's collection.