Hostname: page-component-78c5997874-t5tsf Total loading time: 0 Render date: 2024-11-17T14:58:00.763Z Has data issue: false hasContentIssue false
  • English
  • Français

The pathogenicity for mice of group B streptococci of bovine origin

Published online by Cambridge University Press:  15 May 2009

I. H. Pattison
I. H. Pattison
Affiliation:
Agricultural Research Council Field Station, Compton, Berks
Rights & Permissions [Opens in a new window]

Extract

Core share and HTML view are not available for this content. However, as you have access to this content, a full PDF is available via the ‘Save PDF’ action button.

The following conclusions were drawn from these experiments with bovine group B streptococci:

1. A relationship existed between lethal power and the number of living organisms inoculated, and, therefore, the number of living organisms inoculated must be determined when the killing power of a strain is under investigation.

2. Equal doses of the same strain were likely to kill more mice, but less rapidly, when inoculated intravenously than when inoculated intraperitone-ally.

3. Virulence for mice might be related to serological type.

4. The disease caused by intraperitoneal inoculation of strain S 13 was a rapidly fatal peritonitis with concomitant bacteraemia.

5. The disease caused by intravenous inoculation of strain S13 was a fatal bacteraemia with a tendency for the streptococci to form dense clumps in widely separated tissues.

Type
Research Article
Information
Journal of Hygiene , Volume 47 , Issue 2 , June 1949 , pp. 159 - 165
Copyright
Copyright © Cambridge University Press 1949

References

REFERENCES

Barnard, W. G., & Todd, E. W., (1940). J. Path. Bact. 51, 43.CrossRefGoogle Scholar
Book, M. H., (1933). Amer. J. Path. 9, 569.Google Scholar
Boyd, W., (1938). A Text-book of Pathology. London: Kimpton.CrossRefGoogle Scholar
Brown, J. H., (1939). J. Bact. 37, 133.CrossRefGoogle Scholar
Foster, L. F., (1921). J. Bact. 6, 161.CrossRefGoogle Scholar
Francis, J., & Peters, J. M., (1947). J. Comp. Path. 57, 144.CrossRefGoogle Scholar
Kaplan, M. M., Raiziss, G. W., & Moetsch, J. C., (1942). Amer. J. vet. Res. 3, 392.Google Scholar
Lancefield, R. C., (1934). J. Exp. Med. 59, 441.CrossRefGoogle Scholar
Lancefield, R. C., (1940–1941). Harvey Lect., p. 251.Google Scholar
Little, R. B., & Plastridge, W. N., (1946). Bovine Mastitis. New York and London: McGraw-Hill.Google ScholarPubMed
Mckenzie, D. A., (1941). Vet. Rec. 53, 473.Google Scholar
Mackie, T. J., & McCartney, J. E., (1948). Handbook of Practical Bacteriology. Edinburgh: E. and S. Livingstone.Google Scholar
Minett, F. C., (1935). J. Hyg., Camb., 35, 504.Google Scholar
Minett, F. C., & Stableforth, A. W., (1931). J. Comp. Path. 44, 114.CrossRefGoogle Scholar
Pomales-Lebrón, A., Morales-Otero, P.. & Baralt, J., (1947). Proc. Soc. Exp. Biol, N.Y., 64, 410.CrossRefGoogle Scholar
Sherrington, C. S., (1893). J. Path. Bact. 1, 258.CrossRefGoogle Scholar
Simmons, R. T., & Keogh, E. V., (1940). Aust. J. Exp. Biol. Med. Sci. 18, 151.CrossRefGoogle Scholar
Stableforth, A. W., (1937). J. Path. Bact. 45, 263.CrossRefGoogle Scholar
Todd, E. W., & Hewitt, L. F., (1932). J. Path. Bact. 35, 973.CrossRefGoogle Scholar
Wilson, G. S., & Miles, A. A., (1947). Topley and Wilson's Principles of Bacteriology and Immunity. London: Arnold.Google Scholar