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An Atypical Presentation of Anton Syndrome in a Patient with Preserved Cognition Despite Multiple Cerebral Infarcts: A Case Report

Published online by Cambridge University Press:  07 November 2014

Glen P. Davis ,
R. Andrew Sewell ,
Boaz Levy ,
Bruce H. Price  and
Miles G. Cunningham
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Extract

Visual anosognosia (Anton syndrome) is a rare complication of cortical blindness that results from injury to the visual association cortex, in which patients who are unable to see deny that they are blind. They sometimes confabulate explanations for their visual problems (“there is not enough light to see”), or endanger themselves in efforts to “prove” that they can see (eg, tripping or walking into walls). Without functioning visual-association centers, these patients lack the concept of sight and are unable to acknowledge their impairment. Usually, Anton syndrome is encountered in patients with bilateral occipital infarcts but no other cause for impaired vision. We present an atypical case of Anton syndrome in which the patient's underlying visual deficit was due to advanced glaucoma.

Type
Grand Rounds
Information
CNS Spectrums , Volume 14 , Issue 1 , January 2009 , pp. 15 - 18
Copyright
Copyright © Cambridge University Press 2009

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References

REFERENCES

1.Misra, M, Rath, S, Mohanty, AB. Anton syndrome and cortical blindness due to bilateral occipital infarction. Indian J Ophthalmol. 1989;37:196.Google Scholar
2.Goldenberg, G, Mullbacher, W, Nowak, A. Imagery without perception--a case study of anosognosia for cortical blindness. Neuropsychologia. 1995;33:1373–1382.Google Scholar
3.McDaniel, KD, McDaniel, LD. Anton's syndrome in a patient with posttraumatic optic neuropathy and bifrontal contusions. Arch Neurol. 1991;48:101–105.Google Scholar
4.Folstein, MF, Folstein, SE, McHugh, PR. “Mini-mental state”. A practical method for grading the cognitive state of patients for the clinician. J Psychiatr Res. 1975;12:189–198.Google Scholar
5.McGlynn, SM, Schacter, DL. Unawareness of deficits in neuropsychological syndromes. J Clin Exp Neuropsychol. 1989;2:143–205.CrossRefGoogle Scholar
6.Heilman, KM. Anosognosia: possible neuropsychological mechanisms. In: George, P. Prigatano, GP, Schacter, DL, eds. Awareness of Deficit After Brain Injury: Clinical and Theoretical Issues. New York, NY: Oxford University Press; 1991:53–62.Google Scholar
7.Roca, RP, Klein, LE, Kirby, SM, et al.Recognition of dementia among medical patients. Arch Intern Med. 1984;144:73–75.Google Scholar
8.Garcia, CA, Reding, MJ, Blass, JP. Overdiagnosis of dementia. J Am Geriatr Soc. 1981;29:407–410.Google Scholar
9.Kemp, NM, Brodaty, H, Pond, D, Luscombe, G. Diagnosing dementia in primary care: the accuracy of informant reports. Alzheimer Dis Assoc Disord. 2002;16:171–176.Google Scholar
10.U.S. Preventive Services Task Force. Guide to Clinical Preventive Services. 2nd ed. Washington, DC. U.S. Department of Health and Human Services; 1996.Google Scholar