Book contents
- Frontmatter
- Contents
- List of contributors
- List of abbreviations
- Preface
- Section 1 Bilateral Predominantly Symmetric Abnormalities
- Section 2 Sellar, Perisellar and Midline Lesions
- Section 3 Parenchymal Defects or Abnormal Volume
- Section 4 Abnormalities Without Significant Mass Effect
- Section 5 Primarily Extra-Axial Focal Space-Occupying Lesions
- Section 6 Primarily Intra-Axial Masses
- Section 7 Intracranial Calcifications
- 184 Congenital Cytomegalovirus Infection
- 185 Congenital Toxoplasmosis
- 186 Aicardi–Goutieres Syndrome
- 187 Physiologic Basal Ganglia Calcifications
- 188 Hyperparathyroidism
- 189 Meningioangiomatosis
- 190 Vascular Wall Calcification
- 191 Dystrophic Calcifications
- 192 Calcified Aneurysms
- 193 Vascular Malformations
- 194 Cysticercosis
- 195 Calcified Metastases
- 196 Ganglioglioma
- 197 Oligodendroglioma
- 198 Cortical Tubers in Tuberous Sclerosis
- 199 Subependymal Nodules in Tuberous Sclerosis
- 200 Ependymoma
- 201 Lipoma With Calcification
- 202 Craniopharyngioma
- 203 Meningioma
- 204 Teflon Granuloma
- Index
- References
189 - Meningioangiomatosis
from Section 7 - Intracranial Calcifications
Published online by Cambridge University Press: 05 August 2013
- Frontmatter
- Contents
- List of contributors
- List of abbreviations
- Preface
- Section 1 Bilateral Predominantly Symmetric Abnormalities
- Section 2 Sellar, Perisellar and Midline Lesions
- Section 3 Parenchymal Defects or Abnormal Volume
- Section 4 Abnormalities Without Significant Mass Effect
- Section 5 Primarily Extra-Axial Focal Space-Occupying Lesions
- Section 6 Primarily Intra-Axial Masses
- Section 7 Intracranial Calcifications
- 184 Congenital Cytomegalovirus Infection
- 185 Congenital Toxoplasmosis
- 186 Aicardi–Goutieres Syndrome
- 187 Physiologic Basal Ganglia Calcifications
- 188 Hyperparathyroidism
- 189 Meningioangiomatosis
- 190 Vascular Wall Calcification
- 191 Dystrophic Calcifications
- 192 Calcified Aneurysms
- 193 Vascular Malformations
- 194 Cysticercosis
- 195 Calcified Metastases
- 196 Ganglioglioma
- 197 Oligodendroglioma
- 198 Cortical Tubers in Tuberous Sclerosis
- 199 Subependymal Nodules in Tuberous Sclerosis
- 200 Ependymoma
- 201 Lipoma With Calcification
- 202 Craniopharyngioma
- 203 Meningioma
- 204 Teflon Granuloma
- Index
- References
Summary
Specific Imaging Findings
On non-enhanced CT scans, meningioangiomatosis (MA) may appear as a nodular calcification, as a thickened gyriform mass with or without surrounding hypodense edema, or as hypodense lesion with central calcification. On MRI it is of either low T1 signal, or shows a mixed signal on both T1- and T2-weighted images, depending primarily on the amount of calcific deposition. A pattern of gyriform hyperintensity has been recently reported as a characteristic feature on FLAIR. Contrast enhancement is usually very prominent and may show a gyriform distribution. Surrounding vasogenic edema involving the adjacent white matter is quite common, and the lesion may cause mass effect. Advanced imaging techniques such as MRS may be degraded by contamination artifacts due to calcific deposition; nevertheless, increase in the choline peak with loss of the n-acetyl aspartate peak have been described. Imaging findings are often nonspecific and do not allow for a definite diagnosis.
Pertinent Clinical Information
MA usually affects children and young adults, with a male predominance; it may occur sporadically (most of reported cases) or in association with NF2 (approximately 20%), when it is often multifocal. Sporadic and NF2-associated forms are histologically similar but clinically different. The sporadic type typically presents with partial seizures that are difficult to control, whereas lesions associated with NF2 are often asymptomatic and discovered incidentally. The genetic basis of the sporadic form is not well clarified.
- Type
- Chapter
- Information
- Brain Imaging with MRI and CTAn Image Pattern Approach, pp. 391 - 392Publisher: Cambridge University PressPrint publication year: 2012