To save content items to your account,
please confirm that you agree to abide by our usage policies.
If this is the first time you use this feature, you will be asked to authorise Cambridge Core to connect with your account.
Find out more about saving content to .
To save content items to your Kindle, first ensure email@example.com
is added to your Approved Personal Document E-mail List under your Personal Document Settings
on the Manage Your Content and Devices page of your Amazon account. Then enter the ‘name’ part
of your Kindle email address below.
Find out more about saving to your Kindle.
Note you can select to save to either the @free.kindle.com or @kindle.com variations.
‘@free.kindle.com’ emails are free but can only be saved to your device when it is connected to wi-fi.
‘@kindle.com’ emails can be delivered even when you are not connected to wi-fi, but note that service fees apply.
Genetic analyses which are relevant to plant and animal studies, rather than human populations, have specific limitations in relation to the genetics of schizophrenia. Some reviewers of family studies of schizophrenia have drawn the conclusion that schizophrenia and bipolar disorder do not share the same genetic etiologies whereas others argue that they often do. Considerable effort has been focused on genetic linkage analysis of schizophrenia employing genetic markers in multiply affected families to identify which chromosomal regions harbor susceptibility genes. This approach must take into account the complication of heterogeneity of linkage in which a number of susceptibility genes localized to different chromosomes contribute to the development of schizophrenia. The chapter describes a selection of genes that have been implicated in susceptibility to schizophrenia by cytogenetic, linkage and/or association studies. Twin and adoption studies have shown that the family environment has no influence on the etiology of schizophrenia.
Email your librarian or administrator to recommend adding this to your organisation's collection.