Book chapters will be unavailable on Saturday 24th August between 8am-12pm BST. This is for essential maintenance which will provide improved performance going forwards. Please accept our apologies for any inconvenience caused.
To send content items to your account,
please confirm that you agree to abide by our usage policies.
If this is the first time you use this feature, you will be asked to authorise Cambridge Core to connect with your account.
Find out more about sending content to .
To send content items to your Kindle, first ensure firstname.lastname@example.org
is added to your Approved Personal Document E-mail List under your Personal Document Settings
on the Manage Your Content and Devices page of your Amazon account. Then enter the ‘name’ part
of your Kindle email address below.
Find out more about sending to your Kindle.
Note you can select to send to either the @free.kindle.com or @kindle.com variations.
‘@free.kindle.com’ emails are free but can only be sent to your device when it is connected to wi-fi.
‘@kindle.com’ emails can be delivered even when you are not connected to wi-fi, but note that service fees apply.
Autism, mainly affecting social interaction and verbal and nonverbal communicative functions, is not a single disease, and can be associated with different brain disorders. This chapter focuses on epileptic syndromes or types of epilepsies in which autistic symptoms can be a predominant manifestation, and can worsen or improve in direct correlation with the activity of the epilepsy, even though they probably constitute a minority of situations in which both autism and epilepsy co-occur. The structures of the limbic system thought to be involved in autistic spectrum disorders (ASD) are highly epileptogenic and are the origin of frequently encountered early childhood epilepsies or epileptic syndromes. There are numerous paroxysmal nonepileptic neurological disorders which can be erroneously diagnosed as epilepsy. The clinical diagnosis and management of autistic children and adults, while sharing the same principles as those followed with other handicapped persons, have unique features related to the specific symptoms of ASD.
An action-oriented theory of embodied memory is favorable for many reasons, but it will not provide a quick yet clean solution to the grounding problem in the way Glenberg (1997t) envisages. Although structural mapping via analogical representations may be an adequate mechanism of cognitive representation, it will not suffice to explain representation as such.
To date, neurobiological interest in behaviour and epilepsy has been concerned primarily with temporal lobe epilepsies (TLE), and mesial temporal lobe epilepsies (mTLE) in particular. This concentration on TLE is mostly due to the fact that this type of epilepsy represents the majority of the focal epilepsies – in the Bonn series of patients with pharmacoresistant epilepsies this is about 80% – and that mTLE often forms an entity within the focal epilepsies regarding pathology (hippocampal sclerosis), a frequent history of febrile convulsions, an early onset of epilepsy, and memory problems as the prominent neuropsychological impairment. In TLE, the affected cerebral structures and epileptogenic region are mostly circumscribed, and structural pathology can be well quantified by quantitative MRI (T2 relaxometry and volumetry) or postoperative histopathological examinations of the resected specimen. Frequency, homogeneity and quantifiable pathology provide ideal prerequisites for the study of the functional and behavioural correlates of TLE. Great progress has been made during recent years, at least with respect to the neuropsychological and cognitive aspects of TLE. Recent developments in the field, however, show that it is well recognized that temporal lobe functioning involves more than memory and that its role in emotion and psychiatric symptoms is being rediscovered.
The conditions we meet with frontal lobe epilepsy (FLE) are quite different.
Email your librarian or administrator to recommend adding this to your organisation's collection.