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Cancer of the Esophagus
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  • Cited by 8
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    This book has been cited by the following publications. This list is generated based on data provided by CrossRef.

    Atlabachew, Minaleshewa Chandravanshi, Bhagwan Singh and Redi, Mesfin 2014. Selected Secondary Metabolites and Antioxidant Activity of Khat (Catha edulisForsk) Chewing Leaves Extract. International Journal of Food Properties, Vol. 17, Issue. 1, p. 45.

    Tian, Jianmin and Wang, Kenneth K. 2012. Handbook of Gastrointestinal Cancer. p. 1.

    ., S. Barkwan ., Y. Barnett ., P. Tomkins and ., C.N. Fokunang 2001. Mutagenic and Carcinogenic Activity of Khat (Catha edulis Forsk) Component. Journal of Medical Sciences(Faisalabad), Vol. 1, Issue. 4, p. 276.

    Kassie, Fekadu Darroudi, Firouz Kundi, Michael Schulte-Hermann, Rolf and Knasm�ller, Siegfried 2001. Khat (Catha edulis) consumption causes genotoxic effects in humans. International Journal of Cancer, Vol. 92, Issue. 3, p. 329.

    Ribeiro Pinto, Luis F. Moraes, Emanuela Albano, Rodolpho M. Silva, Maria C. Godoy, Wagner Glisovic, Tina and Lang, Matti A. 2001. Rat oesophageal cytochrome P450 (CYP) monooxygenase system: comparison to the liver and relevance in N-nitrosodiethylamine carcinogenesis. Carcinogenesis, Vol. 22, Issue. 11, p. 1877.

    Haorah, James Miller, Donald W. Brand, Rhonda Smyrk, Thomas C. Wang, Xiaojie Chen, Sheng Chong and Mirvish, Sidney S. 1999. Diffusion of dialkylnitrosamines into the rat esophagus as a factor in esophageal carcinogenesis . Carcinogenesis, Vol. 20, Issue. 5, p. 825.

    POSCHL, HELMUT SEITZ & GUDRUN 1997. INVITED REVIEW Alcohol and gastrointestinal cancer: pathogenic mechanisms. Addiction Biology, Vol. 2, Issue. 1, p. 19.

    Mirvish, Sidney S. 1995. Role of N-nitroso compounds (NOC) and N-nitrosation in etiology of gastric, esophageal, nasopharyngeal and bladder cancer and contribution to cancer of known exposures to NOC. Cancer Letters, Vol. 93, Issue. 1, p. 17.


Book description

The book deals briefly with the normal structure, functioning and biochemistry of the esophagus and with the histological and genetic changes accompanying the development of esophageal cancer in humans and animals. Factors implicated in causing esophageal cancer are described in relation to its very dramatic epidemiology. Thus dietary deficiencies and consumption of foods contaminated by Fusaria mycotoxins are discussed in connection with the extremely high incidence of the disease in certain sharply demarcated regions in China and South Africa, and alcohol and tobacco use are discussed in relation to the epidemiology in Europe and USA. Other hazards mentioned include opium in Iran, betel nut in Asia and bracken in Japan. The sole group of chemicals known to be very potent esophageal carcinogens in animal experiments, the nitrosamines, are described especially in terms of the widespread human exposure.

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